Article
Is environmental carcinogenesis modulated by host polymorphism?
Mutation research - 1 Apr 1991
Idle J R
Abstract excerpt
It is now clear that tobacco smoking, alcohol consumption, dietary factors and occupation can all interact with genetic host factors to place one individual at a greater or lesser risk of a particular cancer than another. Phenotypes which confer significantly elevated risks arise from the human CYP1A1. CYP2D6, GST1 and NAT (N-acetyltransferase) genes. The last is the only one remaining to be cloned. It is...
Read the complete abstract on PubMedTopics
Share this publication in a Topic to start or enrich a Post.
