Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Real-time imaging of de novo arteriovenous malformation in a mouse model of hereditary hemorrhagic telangiectasia.

The Journal of clinical investigation - 1 Nov 2009

Park Sung Ok, Wankhede Mamta, Lee Young Jae, Choi Eun-Jung, Fliess Naime, Choe Se-Woon, Oh Seh-Hoon, Walter Glenn, Raizada Mohan K, Sorg Brian S, Oh S Paul

Abstract excerpt

Arteriovenous malformations (AVMs) are vascular anomalies where arteries and veins are directly connected through a complex, tangled web of abnormal arteries and veins instead of a normal capillary network. AVMs in the brain, lung, and visceral organs, including the liver and gastrointestinal tra...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Activin Receptors, Type I
Share to Activin Receptors, Type II

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Arteriovenous Malformations

Share to Blood Vessels

Share to Diagnostic Imaging

Share to Disease Models, Animal

Share to Homeostasis

Share to Telangiectasia, Hereditary Hemorrhagic

Source

Open in PubMed Open full text

Identifiers

PMID: 19805914
DOI: 10.1172/JCI39482
PMCID: PMC2769195

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-19805914-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Real-time imaging of de novo arteriovenous malformation in a mouse model of hereditary hemorrhagic telangiectasia.PMID 19805914Read ↗

VEGF neutralization can prevent and normalize arteriovenous malformations in an animal model for hereditary hemorrhagic telangiectasia 2.Semantic proximity Read ↗

Mouse models of hereditary hemorrhagic telangiectasia: recent advances and future challengesSemantic proximity Read ↗

A mouse model for hereditary hemorrhagic telangiectasia (HHT) type 2Semantic proximity Read ↗

SMAD4 Deficiency Leads to Development of Arteriovenous Malformations in Neonatal and Adult Mice.Semantic proximity Read ↗

A Microphysiological HHT-on-a-Chip Platform Recapitulates Patient Vascular LesionsSemantic proximity Read ↗

A Microphysiological HHT-on-a-Chip Platform Recapitulates Patient Vascular LesionsSemantic proximity Read ↗

Gene-specific endothelial programs drive AVM pathogenesis in SMAD4 and ALK1 loss-of-functionSemantic proximity Read ↗

Endothelial cell expression of mutant Map2k1 causes vascular malformations in mice.Semantic proximity Read ↗

A non-genetic model of vascular shunts informs on the cellular mechanisms of formation and resolution of arteriovenous malformationsSemantic proximity Read ↗

Arteriovenous malformation in the adult mouse brain resembling the human disease.Semantic proximity Read ↗

Modeling Somatic Second-Hit Mutations in Novel Mouse Models of Hereditary Hemorrhagic TelangiectasiaSemantic proximity Read ↗

Mouse model for hereditary hemorrhagic telangiectasia has a generalized vascular abnormality.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.