Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Complement Receptor 3 Promotes Severe Ross River Virus-Induced Disease

12 Sept 2008

Abstract excerpt

Alphaviruses such as Ross River virus (RRV) and chikungunya virus are mosquito-transmitted viruses that cause explosive epidemics of debilitating arthritis and myositis affecting millions of humans worldwide. Previous studies using a mouse model of RRV-induced disease demonstrated that viral infection results in a severe inflammatory arthritis and myositis and that complement component 3 (C3) contributes to the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Complement system in diseases
Share to Mosquito-borne diseases and control
Share to Viral Infections and Vectors

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 18787004
DOI: 10.1128/jvi.01352-08

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Complement Receptor 3 Promotes Severe Ross River Virus-Induced DiseasePMID 18787004Read ↗

A predominately pulmonary activation of complement in a mouse model of severe COVID-19Semantic proximity Read ↗

Deletion of both the C3a and C5a receptors fails to protect against experimental autoimmune encephalomyelitis.Semantic proximity Read ↗

The Molecular and Cellular Aspects of Arthritis Due to Alphavirus InfectionsSemantic proximity Read ↗

CD8+ T Cells Control Ross River Virus Infection in Musculoskeletal Tissues of Infected MiceSemantic proximity Read ↗

A Factor I-Like Activity Associated with Chikungunya Virus Contributes to Its Resistance to the Human Complement SystemSemantic proximity Read ↗

Loss of TLR3 aggravates CHIKV replication and pathology due to an altered virus-specific neutralizing antibody response.Semantic proximity Read ↗

Complement is primarily activated in the lung in a mouse model of severe COVID-19Semantic proximity Read ↗

Complement C4 Is Protective for Lupus Disease Independent of C3Semantic proximity Read ↗

Targeted disruption of the murine gene coding for the third complement component (C3).Semantic proximity Read ↗

Nonstructural Protein 1 of Variant PEDV Plays a Key Role in Escaping Replication Restriction by Complement C3Semantic proximity Read ↗

Novel Lesions of Bones and Joints Associated with Chikungunya Virus Infection in Two Mouse Models of Disease: New Insights into Disease Pathogenesis.Semantic proximity Read ↗

C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.