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Article

Genistein induces phenotypic reversion of endoglin deficiency in human prostate cancer cells.

Molecular pharmacology - 1 Jan 2008

Craft Clarissa S, Xu Li, Romero Diana, Vary Calvin P H, Bergan Raymond C

Abstract excerpt

Genistein has been shown to inhibit human prostate cancer (PCa) cell motility. Endoglin has been identified as an important suppressor of PCa cell motility, and its expression is lost during PCa progression. It is therefore important to determine whether endoglin loss affects genistein's efficacy and, if so, by what mechanism. In the current study, genistein was shown to induce reversion of endoglin-deficient...

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

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Identifiers

PMID: 17951357
DOI: 10.1124/mol.107.038935

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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