Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Demonstration and Characterization of Mutations Induced by <i>Helicobacter pylori</i> Organisms in Gastric Epithelial Cells

13 Jul 2006

Abstract excerpt

BACKGROUND: Helicobacter pylori gastritis increases gastric cancer risk. Microsatellite instability-type mutations are secondary to deficient DNA mismatch repair. H. pylori gastritis is more frequent in patients with microsatellite instability-positive gastric cancers, and H. pylori organisms independently of inflammation can reduce DNA mismatch repair protein levels, raising the hypothesis that H. pylori...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Digestive system and related health
Share to Genetic factors in colorectal cancer
Share to Helicobacter pylori-related gastroenterology studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 16882331
DOI: 10.1111/j.1523-5378.2006.00408.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Demonstration and Characterization of Mutations Induced by <i>Helicobacter pylori</i> Organisms in Gastric Epithelial CellsPMID 16882331Read ↗

<i>Helicobacter pylori</i>and the molecular pathogenesis of intestinal-type gastric carcinomaSemantic proximity Read ↗

Effect of Helicobacter pylori Infection on the Expression of DNA Mismatch Repair ProteinSemantic proximity Read ↗

Helicobacter pylori genes involved in avoidance of mutations induced by 8-oxoguanine.Semantic proximity Read ↗

Carcinogenic bacterial pathogen <i>Helicobacter pylori</i> triggers DNA double-strand breaks and a DNA damage response in its host cellsSemantic proximity Read ↗

<i>Helicobacter pylori</i> targets cancer-associated apical-junctional constituents in gastroids and gastric epithelial cellsSemantic proximity Read ↗

Pathogen DNA as target for host-generated oxidative stress: role for repair of bacterial DNA damage in Helicobacter pylori colonization.Semantic proximity Read ↗

Strain-specific suppression of <i>microRNA-320</i> by carcinogenic <i>Helicobacter pylori</i> promotes expression of the antiapoptotic protein Mcl-1Semantic proximity Read ↗

Epigenetic Regulation of Tumor Suppressors by<i>Helicobacter pylori</i>Enhances EBV-Induced Proliferation of Gastric Epithelial CellsSemantic proximity Read ↗

CpG island methylator phenotype and Helicobacter pylori infection associated with gastric cancer.Semantic proximity Read ↗

Mosaic DNA imports with interspersions of recipient sequence after natural transformation of Helicobacter pylori.Semantic proximity Read ↗

<i>MGMT</i>and<i>MLH1</i>methylation in<i>Helicobacter pylori</i>-infected children and adultsSemantic proximity Read ↗

Optimized BlaM‐transposon shuttle mutagenesis of <i>Helicobacter pylori</i> allows the identification of novel genetic loci involved in bacterial virulenceSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.