Frequent loss of heterozygosity at 6q in pheochromocytoma.

doi:10.1016/j.humpath.2006.02.002

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Back to search

Article

Frequent loss of heterozygosity at 6q in pheochromocytoma.

Human pathology - 1 Jun 2006

Lemeta Sebsebe, Salmenkivi Kaisa, Pylkkänen Lea, Sainio Markku, Saarikoski Sirkku T, Arola Johanna, Heikkilä Päivi, Haglund Caj, Husgafvel-Pursiainen Kirsti, Böhling Tom

Abstract excerpt

Multiple genetic alterations have been associated with pheochromocytoma (PCC). Most PCCs are sporadic, but they also occur in inherited tumor syndromes, including von Hippel-Lindau disease. Although the etiology of most inherited PCCs is well documented, little is known about the etiology of sporadic tumors. Mutations of those genes that harbor germ-line mutations in familial cases cover only 10% to 15% of...

Topics

Adult
Aged
Alleles
Allelic Imbalance
Cell Cycle Proteins
Chromosomes, Human, Pair 6
DNA, Neoplasm
Female
Gene Deletion
Genetic Markers
Hemangioblastoma
Humans
Loss of Heterozygosity
Male

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

discussions

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Microsatellite Repeats

Middle Aged

Pheochromocytoma

Transcription Factors

Source

Open in PubMed

Identifiers

PMID: 16733217
DOI: 10.1016/j.humpath.2006.02.002

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026.