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Article

Dystrophin is required for appropriate retrograde control of neurotransmitter release at the Drosophila neuromuscular junction.

The Journal of neuroscience : the official journal of the Society for Neuroscience - 4 Jan 2006

van der Plas Mariska C, Pilgram Gonneke S K, Plomp Jaap J, de Jong Anja, Fradkin Lee G, Noordermeer Jasprina N

Abstract excerpt

Mutations in the human dystrophin gene cause the Duchenne and Becker muscular dystrophies. The Dystrophin protein provides a structural link between the muscle cytoskeleton and extracellular matrix to maintain muscle integrity. Recently, Dystrophin has also been found to act as a scaffold for several signaling molecules, but the roles of dystrophin-mediated signaling pathways remain unknown. To further our...

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Identifiers

PMID: 16399704
DOI: 10.1523/JNEUROSCI.4069-05.2006
PMCID: PMC6674336

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Dystrophin is required for appropriate retrograde control of neurotransmitter release at the Drosophila neuromuscular junction.PMID 16399704Read ↗

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