Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Off balance: T-cells in antineutrophil cytoplasmic antibody (ANCA)-associated vasculitides.

Clinical and experimental immunology - 1 Aug 2005

Lamprecht P

Abstract excerpt

There is substantial evidence that T-cells are off balance in antineutrophil cytoplasmic antibody (ANCA)-associated vasculitides. Genetic risk factors may influence shaping of the TCR repertoire and regulatory control of T-cells in predisposed individuals. T-cells are found in inflammatory lesions. Vigorous Th1-type responses are seen in Wegener's granulomatosis and microscopic angiitis, whereas a Th2-type...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antibodies, Antineutrophil Cytoplasmic
Share to Autoimmune Diseases
Share to Cytokines

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to MPO gene

Share to Genetic Predisposition to Disease

Share to T-Lymphocyte Subsets

Share to Vasculitis

Source

Open in PubMed Open full text

Identifiers

PMID: 15996183
DOI: 10.1111/j.1365-2249.2005.02808.x
PMCID: PMC1809434

Genes

MPO

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Off balance: T-cells in antineutrophil cytoplasmic antibody (ANCA)-associated vasculitides.PMID 15996183Read ↗

Environmental factor and inflammation-driven alteration of the total peripheral T-cell compartment in granulomatosis with polyangiitis.Semantic proximity Read ↗

CD8+ T Cells in GCA and GPA: Bystanders or Active Contributors?Semantic proximity Read ↗

T-lymphocytes and disease mechanisms in Wegener's granulomatosis.Semantic proximity Read ↗

In situ detection of PR3-ANCA+ B cells and alterations in the variable region of immunoglobulin genes support a role of inflamed tissue in the emergence of auto-reactivity in granulomatosis with polyangiitisSemantic proximity Read ↗

T helper cell dysfunction in ANCA associated vasculitisSemantic proximity Read ↗

Persistent deficiency of circulating mucosal-associated invariant T (MAIT) cells in ANCA-associated vasculitis.Semantic proximity Read ↗

[Pathophysiology of ANCA-associated vasculitides].Semantic proximity Read ↗

Republished: Antineutrophil cytoplasmic antibody-associated vasculitides: is it time to split up the group?Semantic proximity Read ↗

New insights into the pathogenesis of antineutrophil cytoplasmic autoantibody-associated vasculitis.Semantic proximity Read ↗

Pathogenesis of ANCA-associated vasculitis: new possibilities for intervention.Semantic proximity Read ↗

Update on antiglomerular basement membrane disease.Semantic proximity Read ↗

L20. Memory T-cells in vasculitis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.