Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Alteration of kappa-opioid receptor system expression in distinct brain regions of a genetic model of enhanced ethanol withdrawal severity.

Brain research - 7 Jun 2005

Beadles-Bohling Amy S, Wiren Kristine M

Abstract excerpt

Abrupt withdrawal from chronic alcohol exposure can produce convulsions that are likely due to ethanol (EtOH) neuroadaptations. While significant efforts have focused on elucidating dependence mechanisms, the alterations contributing to EtOH withdrawal severity are less well characterized. The present studies examined the kappa-opioid receptor (KOP-R) system in Withdrawal Seizure-Prone (WSP) and Withdrawal...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Caudate Nucleus
Share to Central Nervous System Depressants

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Enkephalins

Share to Gyrus Cinguli

Share to Nucleus Accumbens

Share to Olfactory Pathways

Share to Putamen

Share to Receptors, Opioid, kappa

Source

Identifiers

PMID: 15869750
DOI: 10.1016/j.brainres.2005.03.043

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Alteration of kappa-opioid receptor system expression in distinct brain regions of a genetic model of enhanced ethanol withdrawal severity.PMID 15869750Read ↗

Chronic ethanol differentially alters susceptibility to chemically induced convulsions in withdrawal seizure-prone and -resistant mice.Semantic proximity Read ↗

Differential expression of GABAA receptor subunit mRNAs in ethanol-naive withdrawal seizure resistant (WSR) vs. withdrawal seizure prone (WSP) mouse brain.Semantic proximity Read ↗

Chronic ethanol treatment alters brain levels of gamma-aminobutyric acidA receptor subunit mRNAs: relationship to genetic differences in ethanol withdrawal seizure severity.Semantic proximity Read ↗

Regulation of operant ethanol-reinforced behavior is genetically independent of regulation of withdrawal severity in WSP and WSR mice.Semantic proximity Read ↗

Kappa Opioid Receptor and Dynorphin Signaling in the Central Amygdala Regulates Alcohol IntakeSemantic proximity Read ↗

Ethanol-regulated gene expression of neuroendocrine specific protein in mice: brain region and genotype specificity.Semantic proximity Read ↗

Potential pleiotropic effects of Mpdz on vulnerability to seizures.Semantic proximity Read ↗

Recapitulating phenotypes of alcohol dependence via overexpression of Oprk1 in the ventral tegmental area of non-dependent TH::Cre rats.Semantic proximity Read ↗

Indexing withdrawal in mice: matching genotypes for exposure in studies using ethanol vapor inhalation.Semantic proximity Read ↗

The ultrasonic vocalization (USV) syllable profile during neonatal opioid withdrawal and a kappa opioid receptor component to increased USV emissions in female miceSemantic proximity Read ↗

Ethanol reward and aversion in mice bred for sensitivity to ethanol withdrawal.Semantic proximity Read ↗

Opioid withdrawal engages a habenular subpopulation responsive to aversive statesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.