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Article

Association study of Wegener granulomatosis and the functionally relevant A645G polymorphism in the bactericidal/permeability increasing protein (BPI) gene.

International journal of immunogenetics - 1 Feb 2005

Jagiello P, Klein W, Schultz H, Csernok E, Gross W L, Epplen J T

Abstract excerpt

In antineutrophil cytoplasmatic antibody (ANCA)-associated vasculitides (AAV), bactericidal/permeability increasing protein (BPI) ANCAs are detected. Recent observations suggest that BPI-ANCAs can potentially contribute to a proinflammatory setting in the absence of proteinase 3 (PRTN3) ANCAs during the development of a pulmonary relapse by impeding the elimination of Gram-negative bacteria (GNB). However, it is...

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Share to Antibodies, Antineutrophil Cytoplasmic
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Share to Enzyme-Linked Immunosorbent Assay

Share to Fluorescent Antibody Technique, Indirect

Share to Granulomatosis with Polyangiitis

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Share to Polymorphism, Restriction Fragment Length

Source

Identifiers

PMID: 15686586
DOI: 10.1111/j.1744-313X.2005.00495.x

Genes

BPI
PRTN3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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