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Article

Relations between eNOS Glu298Asp polymorphism and progression of diabetic nephropathy.

Diabetes research and clinical practice - 1 Sept 2004

Shin Shin Young, Baek Sang Hong, Chang Ki Yuk, Park Cheol Whee, Yang Chul Woo, Jin Dong Chan, Kim Yong Soo, Chang Yoon Sik, Bang Byung Kee

Abstract excerpt

BACKGROUND: Nitric oxide (NO) is related to the pathogenesis of renal hemodynamics in diabetes mellitus. Endothelial nitric oxide synthase (eNOS) gene polymorphism is considered the deterioration factor for progressive renal disease. It has been reported that an interaction of angiotensin II (Ang II) and NO is involved in the control of glomerular hemodynamics. This study aimed to elucidate the roles of eNOS and...

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Share to Diabetes Mellitus, Type 2
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Share to Nitric Oxide Synthase Type III

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Share to Polymorphism, Restriction Fragment Length

Source

Identifiers

PMID: 15331206
DOI: 10.1016/j.diabres.2004.01.010

Genes

ACE

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

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Relations between eNOS Glu298Asp polymorphism and progression of diabetic nephropathy.PMID 15331206Read ↗

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Association between endothelial nitric oxide synthase Glu298Asp gene polymorphism and diabetic nephropathy susceptibility.Semantic proximity Read ↗

Endothelial nitric oxide synthase gene polymorphisms and risk of diabetic nephropathy: a systematic review and meta-analysis.Semantic proximity Read ↗

Interaction of eNOS polymorphism with MTHFR variants increase the risk of diabetic nephropathy and its progression in type 2 diabetes mellitus patients.Semantic proximity Read ↗

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