Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Cellular and ionic basis for the sex-related difference in the manifestation of the Brugada syndrome and progressive conduction disease phenotypes.

Journal of electrocardiology - 1 Jan 2003

Fish Jeffrey M, Antzelevitch Charles

Abstract excerpt

UNLABELLED: The Brugada syndrome (BS) has been linked to mutations in SCN5A. Despite equal hereditary transmission of the mutation between the sexes, the syndrome is 8 to 10 times more likely to occur in males. As recently reported, SCN5A mutations such as G1406R lead to development of BS phenoty...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Arrhythmias, Cardiac
Share to Dogs
Share to Electrocardiography

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to In Vitro Techniques

Share to NAV1.5 Voltage-Gated Sodium Channel

Share to Sex Factors

Share to Sodium Channels

Share to Syndrome

Source

Identifiers

PMID: 14716629
DOI: 10.1016/j.jelectrocard.2003.09.054

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026. Prepared record publication-14716629-2026-09-01.1. Manifest publication-graph-cohort-2026-09-01.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 1 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Cellular and ionic basis for the sex-related difference in the manifestation of the Brugada syndrome and progressive conduction disease phenotypes.PMID 14716629Read ↗

Ionic and cellular basis for the predominance of the Brugada syndrome phenotype in males.Semantic proximity Read ↗

Brugada Syndrome: Clinical, Genetic, Molecular, Cellular, and Ionic Aspects.Semantic proximity Read ↗

Cellular and ionic mechanisms responsible for the Brugada syndrome.Semantic proximity Read ↗

A transient outward potassium current activator recapitulates the electrocardiographic manifestations of Brugada syndrome.Semantic proximity Read ↗

Sex-related differences in incidence, phenotype and risk of sudden cardiac death in inherited arrhythmia syndromesSemantic proximity Read ↗

Ionic and cellular mechanisms underlying the development of acquired Brugada syndrome in patients treated with antidepressants.Semantic proximity Read ↗

Genotype-phenotype correlation in tissue models of Brugada syndrome simulating patients with sodium and calcium channelopathies.Semantic proximity Read ↗

Cellular and ionic basis of J-wave syndromes.Semantic proximity Read ↗

Pathophysiological mechanisms of Brugada syndrome: depolarization disorder, repolarization disorder, or more?Semantic proximity Read ↗

In vivo studies of Scn5a+/- mice modeling Brugada syndrome demonstrate both conduction and repolarization abnormalities.Semantic proximity Read ↗

Differences in sino-atrial and atrio-ventricular function with age and sex attributable to the Scn5a+/- mutation in a murine cardiac model.Semantic proximity Read ↗

Electrophysiological basis and genetics of Brugada syndrome.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.