Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

[Regulation of the expression of the c-fos gene in cells, reverting from being transformed to the pseudonormal phenotype].

Molekuliarnaia biologiia - 1 Jan 2000

Lavrovskiĭ Ia V, Svinarchuk F P, Lavrovskiĭ V A, Konevets D A, Vlasov V V

Abstract excerpt

Regulation of c-fos expression in mice sarcoma cell lines CBA and C3H was investigated. Each of the cell lines was represented by a pair of clones: the tumorigenic and the one, which was produced from it by cloning. It was found, that c-fos expression in cells of the pseudonormal phenotype was similar to that in the normal fibroblasts. Experiments with cells reverted to pseudonormal phenotype transfected...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Base Sequence
Share to Gene Expression Regulation

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Molecular Sequence Data

Share to Phenotype

Share to Plasmids

Share to Promoter Regions, Genetic

Share to RNA, Messenger

Source

Identifiers

PMID: 1435784

Genes

DSE

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

[Regulation of the expression of the c-fos gene in cells, reverting from being transformed to the pseudonormal phenotype].PMID 1435784Read ↗

c-fos gene expression in cell revertants from a transformed to a pseudonormal phenotype.Semantic proximity Read ↗

[Role of the TCF phosphorylation state and the chromatin structure in the negative transcription regulation of the c-fos proto-oncogene in E1A + c-Ha-ras transformed cells].Semantic proximity Read ↗

[The characteristics of the transformed phenotype and the expression of indicator plasmids in the cells of rat embryonic fibroblasts immortalized by oncogene E1Aad5 and transformed by oncogenes E1Aad5+c-Ha-ras].Semantic proximity Read ↗

Mediation of suppression of c-fos transcription in rasT24-transformed rat cells by a cis-acting repressor element.Semantic proximity Read ↗

Cell-type specific multiprotein complex formation over the c-fos serum response element in vivo: ternary complex formation is not required for the induction of c-fos.Semantic proximity Read ↗

A nuclear labile protein, p70, is expressed exclusively during G0-S transition but not in G1 phase of a cell cycle ts mutant, tsJT16.Semantic proximity Read ↗

Effects of mutationally activated Ha-ras on c-fos expression kinetics in rat tracheal epithelial cells.Semantic proximity Read ↗

Lesion-induced increase in nerve growth factor mRNA is mediated by c-fos.Semantic proximity Read ↗

Wild-type murine p53 represses transcription from the murine c-myc promoter in a human glial cell line.Semantic proximity Read ↗

The stimulation of quiescent rat fibroblasts by v-src and v-fps oncogenic protein-tyrosine kinases leads to the induction of a subset of immediate early genes.Semantic proximity Read ↗

Antisense-fos RNA causes partial reversion of the transformed phenotypes induced by the c-Ha-ras oncogene.Semantic proximity Read ↗

Fos expression induces growth arrest in tumorigenic neuroretina cells.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.