Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Cytotoxicity of 125I decay in the DNA double strand break repair deficient mutant cell line, xrs-5.

International journal of radiation biology - 1 Nov 1992

Yasui L S

Abstract excerpt

The survival of parental Chinese hamster ovary (CHO) K1 cells and the DNA double strand break (DSB) repair deficient mutant, xrs-5 was determined after accumulation of 125I decays. Both CHO and xrs-5 cells were extremely sensitive to accumulated 125I decays. The D0 values for CHO and xrs-5 cells were 40 and approximately 7 decays per cell, respectively. The difference in cell survival between CHO and xrs-5 cells...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cell Survival
Share to CHO Cells
Share to Cricetinae

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to DNA Damage

Share to DNA Repair

Share to Iodine Radioisotopes

Share to Mutation

Source

Identifiers

PMID: 1361518
DOI: 10.1080/09553009214552531

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Cytotoxicity of 125I decay in the DNA double strand break repair deficient mutant cell line, xrs-5.PMID 1361518Read ↗

Relationship between PCC fragments and cell killing studied in X-irradiated CHO, CHO-K1 cells and two radiosensitive mutants xrs1 and xrs5.Semantic proximity Read ↗

Reparable and non-reparable DNA strand breaks induced by X-irradiation in CHO K1 cells and the radiosensitive mutants xrs1 and xrs5.Semantic proximity Read ↗

Radiosensitivity throughout the cell cycle and repair of potentially lethal damage and DNA double-strand breaks in an X-ray-sensitive CHO mutant.Semantic proximity Read ↗

Radiosensitivity, apoptosis and repair of DNA double-strand breaks in radiation-sensitive Chinese hamster ovary cell mutants treated at different dose rates.Semantic proximity Read ↗

Recovery of the radiation survival-curve shoulder in CHO-KI, XRS-5 and revertant XRS-5 populations.Semantic proximity Read ↗

Enhanced mutability at the tk locus in the radiosensitive double-strand break repair mutant xrs5.Semantic proximity Read ↗

A DNA double-strand break repair-deficient mutant of CHO cells shows reduced radiosensitization after exposure to hyperthermic temperatures in the plateau phase of growth.Semantic proximity Read ↗

Efficient mutation induction by 125I and 131I decays in DNA of human cells.Semantic proximity Read ↗

Changes in the nuclear structure in the radiation-sensitive CHO mutant cell, xrs-5.Semantic proximity Read ↗

Response of X-ray-sensitive CHO mutant cells (xrs-6c) to radiation. II. Relationship between cell survival and the induction of chromosomal damage with low doses of alpha particles.Semantic proximity Read ↗

An ionizing radiation-sensitive mutant of CHO cells: irs-20. II. Dose-rate effects and cellular recovery processes.Semantic proximity Read ↗

The induction and reversal of topoisomerase II cleavable complexes formed by nuclear extract from the CHO DNA repair mutant, xrs1.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.