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Article

Connexin26-mediated gap junctional communication reverses the malignant phenotype of MCF-7 breast cancer cells.

Cancer science - 1 Jun 2003

Momiyama Megumi, Omori Yasufumi, Ishizaki Yasuko, Nishikawa Yuji, Tokairin Takuo, Ogawa Jun-ichi, Enomoto Katsuhiko

Abstract excerpt

A growing body of evidence indicates that the gap junction (GJ) plays a pivotal role in tumor suppression by exerting cell-cell communication. It has, however, been reported that expression of connexin26 (Cx26) protein is induced in human ductal carcinomas of the breast and that its amount increa...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Source

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Identifiers

PMID: 12824874
DOI: 10.1111/j.1349-7006.2003.tb01473.x
PMCID: PMC11160258

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-12824874-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

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Connexin26-mediated gap junctional communication reverses the malignant phenotype of MCF-7 breast cancer cells.PMID 12824874Read ↗

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