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Article

Ptprj is a candidate for the mouse colon-cancer susceptibility locus Scc1 and is frequently deleted in human cancers

24 Jun 2002

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Share to Genetic factors in colorectal cancer
Share to Protein Tyrosine Phosphatases
Share to RNA modifications and cancer

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 12089527
DOI: 10.1038/ng903

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Ptprj is a candidate for the mouse colon-cancer susceptibility locus Scc1 and is frequently deleted in human cancersPMID 12089527Read ↗

PTPRJ haplotypes and colorectal cancer risk.Semantic proximity Read ↗

Genetic ablation of Ptprj, a mouse cancer susceptibility gene, results in normal growth and development and does not predispose to spontaneous tumorigenesis.Semantic proximity Read ↗

Allelic association of the human homologue of the mouse modifier Ptprj with breast cancer.Semantic proximity Read ↗

Clustering of colon, lung, and other cancer susceptibility genes with protein tyrosine phosphatases and protein kinases in multiple short genomic regionsSemantic proximity Read ↗

Missense polymorphisms of PTPRJ and PTPN13 genes affect susceptibility to a variety of human cancers.Semantic proximity Read ↗

Genomic Sequencing of Key Genes in Mouse Pancreatic Cancer CellsSemantic proximity Read ↗

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MicroRNA genes are frequently located near mouse cancer susceptibility loci.Semantic proximity Read ↗

10 years of mouse cancer modifier loci: human relevance.Semantic proximity Read ↗

Genome-wide association and fine mapping of genetic loci predisposing to colon carcinogenesis in mice.Semantic proximity Read ↗

G1 checkpoint failure and increased tumor susceptibility in mice lacking the novel p53 target Ptprv.Semantic proximity Read ↗

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