Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Apparent replication of suggestive linkage on chromosome 16 in the NIMH genetics initiative bipolar pedigrees.

American journal of medical genetics - 8 May 2002

Dick Danielle M, Foroud Tatiana, Edenberg Howard J, Miller Marvin, Bowman Elizabeth, Rau N Leela, DePaulo J Raymond, McInnis Melvin, Gershon Elliot, McMahon Francis, Rice John P, Bierut Laura J, Reich Theodore, Nurnberger John

Abstract excerpt

Analyses of a replication sample of families collected as part of the National Institute of Mental Health (NIMH) Genetics Initiative for bipolar disorder provide further evidence for linkage to a region of chromosome 16. Families who had a bipolar I (BPI) proband and at least one BPI or schizoaffective, bipolar type (SABP) first-degree relative were ascertained for the purpose of identifying genes involved in...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bipolar Disorder
Share to Chromosome Mapping

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Chromosomes, Human, Pair 16

Share to BPI gene

Share to Genetic Linkage

Share to Genetic Predisposition to Disease

Share to Lod Score

Share to Pedigree

Source

Identifiers

PMID: 11992562
DOI: 10.1002/ajmg.10380

Genes

BPI

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Apparent replication of suggestive linkage on chromosome 16 in the NIMH genetics initiative bipolar pedigrees.PMID 11992562Read ↗

Further evidence for linkage of bipolar disorder to chromosomes 6 and 17 in a new independent pedigree series.Semantic proximity Read ↗

Initial genomic scan of the NIMH genetics initiative bipolar pedigrees: chromosomes 3, 5, 15, 16, 17, and 22.Semantic proximity Read ↗

Genome-wide parametric linkage analyses of 644 bipolar pedigrees suggest susceptibility loci at chromosomes 16 and 20.Semantic proximity Read ↗

Genome-wide scan and conditional analysis in bipolar disorder: evidence for genomic interaction in the National Institute of Mental Health genetics initiative bipolar pedigrees.Semantic proximity Read ↗

Strategies to identify genes for complex disorders: a focus on bipolar disorder and chromosome 16p.Semantic proximity Read ↗

Evidence of susceptibility loci on 4q32 and 16p12 for bipolar disorder.Semantic proximity Read ↗

Linkage analysis in psychiatric disorders: the emerging picture.Semantic proximity Read ↗

Linkage disequilibrium mapping of the chromosome 6q21-22.31 bipolar I disorder susceptibility locus.Semantic proximity Read ↗

Genome-wide scan of bipolar disorder and investigation of population stratification effects on linkage: support for susceptibility loci at 4q21, 7q36, 9p21, 12q24, 14q24, and 16p13.Semantic proximity Read ↗

Evidence for a putative bipolar disorder locus on 2p13-16 and other potential loci on 4q31, 7q34, 8q13, 9q31, 10q21-24, 13q32, 14q21 and 17q11-12.Semantic proximity Read ↗

Genome-wide scan of bipolar disorder in 65 pedigrees: supportive evidence for linkage at 8q24, 18q22, 4q32, 2p12, and 13q12.Semantic proximity Read ↗

Loci on chromosomes 6q and 6p interact to increase susceptibility to bipolar affective disorder in the national institute of mental health genetics initiative pedigrees.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.