Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Selective inhibition of splicing at the avian sarcoma virus src 3' splice site by direct-repeat posttranscriptional cis elements.

Journal of virology - 1 Sept 2000

Guo W, Winistorfer S C, Stoltzfus C M

Abstract excerpt

The direct-repeat elements (dr1) of avian sarcoma virus (ASV) and leukosis virus have the properties of constitutive transport elements (CTEs), which facilitate cytoplasmic accumulation of unspliced RNA. It is thought that these elements represent binding sites for cellular factors. Previous studies have indicated that in the context of the avian sarcoma virus genome, precise deletion of both ASV dr1 elements...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Avian Sarcoma Viruses
Share to Chick Embryo
Share to Fibroblasts

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Genes, src

Share to Regulatory Sequences, Nucleic Acid

Share to Repetitive Sequences, Nucleic Acid

Share to RNA Splicing

Share to Virus Replication

Source

Open in PubMed Open full text

Identifiers

PMID: 10954552
DOI: 10.1128/jvi.74.18.8513-8523.2000
PMCID: PMC116363

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Selective inhibition of splicing at the avian sarcoma virus src 3' splice site by direct-repeat posttranscriptional cis elements.PMID 10954552Read ↗

Selection and characterization of replication-competent revertants of a Rous sarcoma virus src gene oversplicing mutant.Semantic proximity Read ↗

Rous sarcoma virus direct repeat cis elements exert effects at several points in the virus life cycle.Semantic proximity Read ↗

Mutations in the regions of the Rous sarcoma virus 3' splice sites: implications for regulation of alternative splicing.Semantic proximity Read ↗

Rous Sarcoma Virus DR Posttranscriptional Elements Use a Novel RNA Export PathwaySemantic proximity Read ↗

The role of branchpoint and 3'-exon sequences in the control of balanced splicing of avian retrovirus RNA.Semantic proximity Read ↗

Genetic selection for balanced retroviral splicing: novel regulation involving the second step can be mediated by transitions in the polypyrimidine tract.Semantic proximity Read ↗

Inhibition of RNA splicing is a novel therapeutic strategy for disruption of nuclear replicating virusesSemantic proximity Read ↗

The upstream, direct repeat sequence of Prague A Rous sarcoma virus is deficient in mediating efficient Gag assembly and particle release.Semantic proximity Read ↗

Does steric interference between splice sites block the splicing of a short c-src neuron-specific exon in non-neuronal cells?Semantic proximity Read ↗

Control of retroviral RNA splicing through maintenance of suboptimal processing signals.Semantic proximity Read ↗

Efficient polyadenylation of Rous sarcoma virus RNA requires the negative regulator of splicing element.Semantic proximity Read ↗

Transformation-defective mutants with 5' deletions of the src gene are frequently generated during replication of Rous sarcoma virus in established quail fibroblasts.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.