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Tubular overexpression of gremlin in transgenic mice aggravates renal damage in diabetic nephropathyDOI 10.7490/f1000research.1098222.1
Tubular overexpression of gremlin induces renal damage susceptibility in mice.Semantic proximity
Gremlin1 plays a key role in kidney development and renal fibrosis.Semantic proximity
Suppression of Gremlin by RNA interference attenuates glucose-induced fibrogenesis in rat mesangial cellsSemantic proximity
Allelic Depletion of <i>grem1</i> Attenuates Diabetic Kidney DiseaseSemantic proximity
PGC1-α in diabetic kidney disease: unraveling renoprotection and molecular mechanismsSemantic proximity
Co-regulation of Gremlin and Notch signalling in diabetic nephropathySemantic proximity
Diabetic kidney lesions of GIPRdn transgenic mice: podocyte hypertrophy and thickening of the GBM precede glomerular hypertrophy and glomerulosclerosis.Semantic proximity
Insights into the Role of Gremlin-1, a Bone Morphogenic Protein Antagonist, in Cancer Initiation and ProgressionSemantic proximity
Gremlin: an example of the re-emergence of developmental programmes in diabetic nephropathySemantic proximity
Advances in Murine Models of Diabetic NephropathySemantic proximity
Disruption of tubular Flcn expression as a mouse model for renal tumor inductionSemantic proximity
Klotho attenuates renal hypertrophy and glomerular injury in Ins2Akita diabetic mice.Semantic proximity
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