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TIMELESS promotes the proliferation and migration of lung adenocarcinoma cells by activating EGFR through AMPK and SPHK1 regulation

2022-11-01

Abstract excerpt

<h4>Background: </h4> Lung adenocarcinoma (LUAD) has high morbidity and is prone to recurrence. TIMELESS (TIM), which regulates circadian rhythms in Drosophila, is highly expressed in various tumors. <h4>Methods: </h4>: We used tumor samples from patients with lung carcinoma and LUAD patient data from public databases to confirm the relationship of TIM expression with lung cancer. We used NSCLC cell lines and siR...

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Literature Corpus work
a698e5e4-a920-5158-a04d-9ad4d893cddd
DOI
10.21203/rs.3.rs-2157503/v1
Open publication

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TIMELESS promotes the proliferation and migration of lung adenocarcinoma cells by activating EGFR through AMPK and SPHK1 regulationDOI 10.21203/rs.3.rs-2157503/v1
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