Article
High-Plex CSF proteomics reveals a distinct vascular- immune signature in β-amyloid-negative corticobasal syndrome
2026-05-28
Abstract excerpt
<title>Abstract</title> <p>Background The clinical phenotype of corticobasal syndrome can be caused by aggregation of various proteins in the brain, including 4-repeat tau in a substantial proportion of the patients. Reliable biomarkers for differentiating corticobasal syndrome with aggregation of 4-repeat tau from related parkinsonian disorders remain limited. We therefore aimed to (i) map syndrome-specific CSF...
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Identifiers and source
- Literature Corpus work
- 3ac0e873-a869-52b4-a525-256cf39e3227
- DOI
- 10.21203/rs.3.rs-9437991/v1