Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Autoimmune orchitis, epididymitis, and vasitis are immunogenetically distinct lesions.

The American journal of pathology - 1 May 1998

Roper R J, Doerge R W, Call S B, Tung K S, Hickey W F, Teuscher C

Abstract excerpt

Experimental allergic orchitis (EAO), the principle animal model of noninfectious testicular inflammatory disease, is a genetically determined phenotype. Classical EAO, induced by inoculation with testicular homogenate and the appropriate adjuvants, is characterized by inflammatory infiltrates in the testis (orchitis), epididymis (epididymitis), and vas deferens (vasitis). In this study, the genetic control of...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Chromosome Mapping
Share to DNA
Share to Genetic Linkage

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Genetic Predisposition to Disease

Share to Genotype

Share to Mice, Inbred Strains

Share to Polymorphism, Genetic

Source

Open in PubMed Open full text

Identifiers

PMID: 9588902
PMCID: PMC1858582

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Autoimmune orchitis, epididymitis, and vasitis are immunogenetically distinct lesions.PMID 9588902Read ↗

Experimental allergic orchitis in mice. VI. Recombinations within the H-2S/H-2D interval define the map position of the H-2-associated locus controlling disease susceptibility.Semantic proximity Read ↗

Genome-wide search for genes that modulate inflammatory arthritis caused by Ali18 mutation in mice.Semantic proximity Read ↗

Genetic analysis of disease subtypes and sexual dimorphisms in mouse experimental allergic encephalomyelitis (EAE): relapsing/remitting and monophasic remitting/nonrelapsing EAE are immunogenetically distinct.Semantic proximity Read ↗

Identification of Orch3, a locus controlling dominant resistance to autoimmune orchitis, as kinesin family member 1C.Semantic proximity Read ↗

Aod1 controlling day 3 thymectomy-induced autoimmune ovarian dysgenesis in mice encompasses two linked quantitative trait loci with opposing allelic effects on disease susceptibility.Semantic proximity Read ↗

Genetics of experimental allergic encephalomyelitis supports the role of T helper cells in multiple sclerosis pathogenesis.Semantic proximity Read ↗

Multiple loci govern the bone marrow-derived immunoregulatory mechanism controlling dominant resistance to autoimmune orchitis.Semantic proximity Read ↗

ENU-mutagenesis: insight into immune function and pathology.Semantic proximity Read ↗

Genome-wide and species-wide dissection of the genetics of arthritis severity in heterogeneous stock mice.Semantic proximity Read ↗

Transcriptomics analysis of allergen-induced inflammatory gene expression in the Four-Core Genotype mouse model.Semantic proximity Read ↗

Resolution of a 16.8-Mb autoimmunity-regulating rat chromosome 4 region into multiple encephalomyelitis quantitative trait loci and evidence for epistasis.Semantic proximity Read ↗

Interacting quantitative trait loci control loss of peripheral tolerance and susceptibility to autoimmune ovarian dysgenesis after day 3 thymectomy in mice.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.