Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Decreased intracellular survival of an fkpA mutant of Salmonella typhimurium Copenhagen.

Infection and immunity - 1 Feb 1997

Horne S M, Kottom T J, Nolan L K, Young K D

Abstract excerpt

The fkpA gene of Salmonella typhimurium encodes a protein similar to the macrophage infectivity potentiator (Mip) proteins of Legionella pneumophila and Chlamydia trachomatis. Because Mip proteins enhance the ability of these intracellular pathogens to survive within macrophages and epithelial cells, we tested whether the product of the fkpA gene would have the same effect on the intracellular growth of a...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bacterial Proteins
Share to Genes, Bacterial
Share to Membrane Proteins

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Mutagenesis

Share to Phenotype

Share to Salmonella typhimurium

Share to Virulence

Source

Open in PubMed Open full text

Identifiers

PMID: 9009347
DOI: 10.1128/iai.65.2.806-810.1997
PMCID: PMC176130

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Decreased intracellular survival of an fkpA mutant of Salmonella typhimurium Copenhagen.PMID 9009347Read ↗

The Francisella tularensis LVS ΔpdpC mutant exhibits a unique phenotype during intracellular infection.Semantic proximity Read ↗

Characterization of a Salmonella choleraesuis mutant that cannot multiply within epithelial cells.Semantic proximity Read ↗

Intracellular replication is essential for the virulence of Salmonella typhimurium.Semantic proximity Read ↗

Role of periplasmic peptidylprolyl isomerases in Salmonella enterica serovar Typhimurium virulence.Semantic proximity Read ↗

A Burkholderia pseudomallei Macrophage Infectivity Potentiator-Like Protein Has Rapamycin-Inhibitable Peptidylprolyl Isomerase Activity and Pleiotropic Effects on VirulenceSemantic proximity Read ↗

Survival of Enterococcus faecalis in mouse peritoneal macrophages.Semantic proximity Read ↗

Trafficking of porin-deficient Salmonella typhimurium mutants inside HeLa cells: ompR and envZ mutants are defective for the formation of Salmonella-induced filaments.Semantic proximity Read ↗

Synergistic effect of mutations in invA and lpfC on the ability of Salmonella typhimurium to cause murine typhoid.Semantic proximity Read ↗

Salmonella typhi rpoS mutant is less cytotoxic than the parent strain but survives inside resting THP-1 macrophages.Semantic proximity Read ↗

Biochemical and functional analyses of the Mip protein: influence of the N-terminal half and of peptidylprolyl isomerase activity on the virulence of Legionella pneumophila.Semantic proximity Read ↗

Salmonella typhimurium thyA mutants fail to grow intracellularly in vitro and are attenuated in mice.Semantic proximity Read ↗

dksA is required for intercellular spread of Shigella flexneri via an RpoS-independent mechanism.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.