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Article

Defective propagation of signals generated by sympathetic nerve stimulation in the liver of connexin32-deficient mice.

Proceedings of the National Academy of Sciences of the United States of America - 3 Sept 1996

Nelles E, Bützler C, Jung D, Temme A, Gabriel H D, Dahl U, Traub O, Stümpel F, Jungermann K, Zielasek J, Toyka K V, Dermietzel R, Willecke K

Abstract excerpt

The gap junctional protein connexin32 is expressed in hepatocytes, exocrine pancreatic cells, Schwann cells, and other cell types. We have inactivated the connexin32 gene by homologous recombination in the mouse genome and have generated homozygous connexin32-deficient mice that were viable and fertile but weighed on the average approximately 17% less than wild-type controls. Electrical stimulation of sympathetic...

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Identifiers

PMID: 8790370
DOI: 10.1073/pnas.93.18.9565
PMCID: PMC38468

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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