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Article

Promoter-specific trans-activation and inhibition mediated by JunB.

Cancer research - 15 Aug 1993

Hsu J C, Cressman D E, Taub R

Abstract excerpt

Nuclear levels of c-Jun, JunB, c-Fos, and LRF-1 (liver regeneration factor) are high for a large fraction of the G1 phase in regenerating liver and mitogen-stimulated hepatic cells. Previously, JunB was regarded as a less potent transcriptional activator than c-Jun that could also function as a r...

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Identifiers

PMID: 8339292

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-8339292-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

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Promoter-specific trans-activation and inhibition mediated by JunB.PMID 8339292Read ↗

Interactions among LRF-1, JunB, c-Jun, and c-Fos define a regulatory program in the G1 phase of liver regeneration.Semantic proximity Read ↗

JunB can substitute for Jun in mouse development and cell proliferation.Semantic proximity Read ↗

Induction of the AP-1 members c-Jun and JunB by TGF-β/Smad suppresses early Smad-driven gene activationSemantic proximity Read ↗

JunB contributes to Id2 repression and the epithelial–mesenchymal transition in response to transforming growth factor–βSemantic proximity Read ↗

Both Jun and Fos contribute to transcription activation by the heterodimer.Semantic proximity Read ↗

Chronic myeloid leukemia with increased granulocyte progenitors in mice lacking junB expression in the myeloid lineage.Semantic proximity Read ↗

JUN-CCAAT/enhancer-binding protein complexes inhibit surfactant-associated protein B promoter activity.Semantic proximity Read ↗

Identification and Functional Characterization of a Smad Binding Element (SBE) in the JunB Promoter That Acts as a Transforming Growth Factor-β, Activin, and Bone Morphogenetic Protein-inducible EnhancerSemantic proximity Read ↗

Defective endochondral ossification in mice with strongly compromised expression of JunB.Semantic proximity Read ↗

JunB gene expression is inactivated by methylation in chronic myeloid leukemiaSemantic proximity Read ↗

Constitutive JunB expression, associated with the JAK2 V617F mutation, stimulates proliferation of the erythroid lineageSemantic proximity Read ↗

Transcriptional Regulatory Signals Define Cytokine-Dependent and -Independent Pathways in Liver RegenerationSemantic proximity Read ↗

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