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Article

Defective endochondral ossification in mice with strongly compromised expression of JunB.

Journal of cell science - 15 Nov 2003

Hess Jochen, Hartenstein Bettina, Teurich Sibylle, Schmidt Dirk, Schorpp-Kistner Marina, Angel Peter

Abstract excerpt

Functional analysis in mice has established an absolute requirement of JunB, a member of the AP-1 transcription factor family, during early embryonic development. To investigate the role of JunB during mid and late gestation and postnatally Ubi-junB transgenic mice were used to generate two junB-...

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Source

Identifiers

PMID: 14576352
DOI: 10.1242/jcs.00772

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-14576352-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

Defective endochondral ossification in mice with strongly compromised expression of JunB.PMID 14576352Read ↗

Mice lacking JunB are osteopenic due to cell-autonomous osteoblast and osteoclast defects.Semantic proximity Read ↗

JunB can substitute for Jun in mouse development and cell proliferation.Semantic proximity Read ↗

JunB is essential for mammalian placentation.Semantic proximity Read ↗

Chronic myeloid leukemia with increased granulocyte progenitors in mice lacking junB expression in the myeloid lineage.Semantic proximity Read ↗

JunB contributes to Id2 repression and the epithelial–mesenchymal transition in response to transforming growth factor–βSemantic proximity Read ↗

Expansion of Bone Precursors through Jun as a Novel Treatment for Osteoporosis-Associated Fractures.Semantic proximity Read ↗

JunB inhibits proliferation and transformation in B-lymphoid cellsSemantic proximity Read ↗

Targeted disruption of the murine junD gene results in multiple defects in male reproductive function.Semantic proximity Read ↗

Identification and Functional Characterization of a Smad Binding Element (SBE) in the JunB Promoter That Acts as a Transforming Growth Factor-β, Activin, and Bone Morphogenetic Protein-inducible EnhancerSemantic proximity Read ↗

c-Jun N-Terminal Kinases (JNKs) Are Critical Mediators of Osteoblast Activity In VivoSemantic proximity Read ↗

JunB defines functional and structural integrity of the epidermo-pilosebaceous unit in the skinSemantic proximity Read ↗

Expression of FosB during mouse development: normal development of FosB knockout mice.Semantic proximity Read ↗

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