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Article

Induction of the AP-1 members c-Jun and JunB by TGF-β/Smad suppresses early Smad-driven gene activation

26 Apr 2001

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Share to Cancer-related gene regulation
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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 11402315
DOI: 10.1038/sj.onc.1204347

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

Induction of the AP-1 members c-Jun and JunB by TGF-β/Smad suppresses early Smad-driven gene activationPMID 11402315Read ↗

Smad3 and Smad4 cooperate with c-Jun/c-Fos to mediate TGF-β-induced transcriptionSemantic proximity Read ↗

Smad3/AP-1 interactions control transcriptional responses to TGF-β in a promoter-specific mannerSemantic proximity Read ↗

TGFβ inducible early gene enhances TGFβ/Smad-dependent transcriptional responsesSemantic proximity Read ↗

Smads bind directly to the Jun family of AP-1 transcription factorsSemantic proximity Read ↗

Transcriptional Activators of TGF-β Responses: SmadsSemantic proximity Read ↗

Bone development and inflammatory disease is regulated by AP-1 (Fos/Jun)Semantic proximity Read ↗

Smad3 mediates immediate early induction of Id1 by TGF-βSemantic proximity Read ↗

Identification and Functional Characterization of a Smad Binding Element (SBE) in the JunB Promoter That Acts as a Transforming Growth Factor-β, Activin, and Bone Morphogenetic Protein-inducible EnhancerSemantic proximity Read ↗

SMADs: mediators and regulators of TGF-β signalingSemantic proximity Read ↗

Interdependent SMAD and JNK Signaling in Transforming Growth Factor-β-mediated TranscriptionSemantic proximity Read ↗

Specific interactions between Smad proteins and AP-1 components determine TGFβ-induced breast cancer cell invasionSemantic proximity Read ↗

Critical role of Smads and AP-1 complex in transforming growth factor-beta -dependent apoptosis.Semantic proximity Read ↗

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