Int-6, a highly conserved, widely expressed gene, is mutated by mouse mammary tumor virus in mammary preneoplasia.

doi:10.1128/JVI.69.3.1932-1938.1995

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Back to search

Article

Int-6, a highly conserved, widely expressed gene, is mutated by mouse mammary tumor virus in mammary preneoplasia.

Journal of virology - 1 Mar 1995

Marchetti A, Buttitta F, Miyazaki S, Gallahan D, Smith G H, Callahan R

Abstract excerpt

With a unique mouse mammary tumor model system in which mouse mammary tumor virus (MMTV) insertional mutations can be detected during progression from preneoplasia to frank malignancy, including metastasis, we have discovered a new common integration site (designated Int-6) for MMTV in mouse mamm...

Topics

Amino Acid Sequence
Animals
Base Sequence
Cell Transformation, Viral
Cloning, Molecular
Eukaryotic Initiation Factor-3
Gene Expression Regulation, Viral
Genes
Mammary Neoplasms, Experimental
Mammary Tumor Virus, Mouse
Mice
Molecular Sequence Data
Mutagenesis, Insertional
Mutation
Precancerous Conditions
Proto-Oncogene Proteins
Proviruses
RNA, Messenger

Source

Open in PubMed Open full text

Identifiers

PMID: 7853537
DOI: 10.1128/JVI.69.3.1932-1938.1995
PMCID: PMC188811

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026. Prepared record publication-7853537-2026-08-30.1. Manifest publication-graph-cohort-2026-08-30.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 30 Aug 2026.

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

discussions

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics