Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Specific Interaction of Escherichia coli 0157:H7-Derived Shiga-like Toxin II with Human Renal Endothelial Cells

1 Nov 1995

Abstract excerpt

In Escherichia coli O157:H7 foodborne infections of humans, the Shiga-like toxins (SLTs) are thought to be the cause of life-threatening vascular complications, including acute renal disease known as hemolytic uremic syndrome or HUS. As virtually all E. coli O157:H7 isolates from HUS patients produce SLT-II (vs. SLT-I), the possible preferential interaction of SLT-II with human renal microvascular endothelial...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Escherichia coli research studies
Share to Vibrio bacteria research studies
Share to Viral gastroenteritis research and epidemiology

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 7594687
DOI: 10.1093/infdis/172.5.1397

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Specific Interaction of Escherichia coli 0157:H7-Derived Shiga-like Toxin II with Human Renal Endothelial CellsPMID 7594687Read ↗

Molecular Biology of Escherichia coli Shiga Toxins’ Effects on Mammalian CellsSemantic proximity Read ↗

The pathogenic mechanisms of Shiga toxin and the Shiga‐like toxinsSemantic proximity Read ↗

Molecular and functional analysis of Shiga toxin–induced response patterns in human vascular endothelial cellsSemantic proximity Read ↗

Synergy of LPS and Shiga-toxin exposure on the RNA expression program of cultured human umbilical vein endothelial cellsSemantic proximity Read ↗

Shiga toxin-associated hemolytic uremic syndrome: advances in pathogenesis and therapeutics.Semantic proximity Read ↗

New Aspects in the Pathogenesis of Enteropathic Hemolytic Uremic SyndromeSemantic proximity Read ↗

Genetic Susceptibility to Postdiarrheal Hemolytic-Uremic Syndrome After Shiga Toxin-Producing Escherichia coli Infection: A Centers for Disease Control and Prevention FoodNet Study.Semantic proximity Read ↗

Pathogenesis of Shiga Toxin-Associated Hemolytic Uremic SyndromeSemantic proximity Read ↗

Cytolethal distending toxin in Escherichia coli O157:H7: spectrum of conservation, structure, and endothelial toxicity.Semantic proximity Read ↗

Variants of Shiga-like toxin II constitute a major toxin component in Escherichia coli O157 strains from patients with haemolytic uraemic syndrome.Semantic proximity Read ↗

Heme as Possible Contributing Factor in the Evolvement of Shiga-Toxin Escherichia coli Induced Hemolytic-Uremic Syndrome.Semantic proximity Read ↗

Distinct renal pathology and a chemotactic phenotype after enterohemorrhagic Escherichia coli shiga toxins in non-human primate models of hemolytic uremic syndrome.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.