Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Enhancing throughput and robustness of the fibroblast to myofibroblast transition assay

14 Mar 2025

Abstract excerpt

Idiopathic pulmonary fibrosis (IPF) is a chronic and progressive age-related lung disease with an average survival of 3-5 years post-diagnosis if left untreated. It is characterized by lung fibrosis, inflammation, and destruction of lung architecture, leading to worsening respiratory symptoms and physiological impairment, ultimately culminating in progressive respiratory failure. The development of novel...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis
Share to Medical Imaging and Pathology Studies
Share to Peptidase Inhibition and Analysis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 40090552
DOI: 10.1016/j.slasd.2025.100226

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Enhancing throughput and robustness of the fibroblast to myofibroblast transition assayPMID 40090552Read ↗

Modeling cell-cell interactions to advance drug discovery in Idiopathic Pulmonary FibrosisSemantic proximity Read ↗

The Scar-in-a-Jar: In Vitro Fibrosis Model for Anti-Fibrotic Drug Testing.Semantic proximity Read ↗

Phenotypic Drug Screening for Novel Antifibrotic Therapeutics in Lung Health.Semantic proximity Read ↗

Fibrotic human lung extracellular matrix as a disease-specific substrate for 3D <i>in-vitro</i> models of pulmonary fibrosisSemantic proximity Read ↗

Designing and utilising an in vitro model to investigate cellular pathways involved in idiopathic pulmonary fibrosis and connective tissue disease associated interstitial lung diseaseSemantic proximity Read ↗

Engineering a 3D Lung Co-culture Platform to Model Epithelial–Fibroblast Interactions in Pulmonary FibrosisSemantic proximity Read ↗

FLECS Technology for High-Throughput Screening of Hypercontractile Cellular Phenotypes in Fibrosis: A Function-First Approach to Anti-Fibrotic Drug DiscoverySemantic proximity Read ↗

FLECS technology for high-throughput screening of hypercontractile cellular phenotypes in fibrosis: A function-first approach to anti-fibrotic drug discovery.Semantic proximity Read ↗

Highlighting fibroblast plasticity in lung fibrosis: the WI-38 cell line as a model for investigating the myofibroblast and lipofibroblast switchSemantic proximity Read ↗

Highlighting fibroblast plasticity in lung fibrosis: the WI-38 cell line as a model for investigating the myofibroblast and lipofibroblast switch.Semantic proximity Read ↗

Multiomic and quantitative label-free microscopy-based analysis of <i>ex vivo</i> culture and TGFbeta1 stimulation of human precision-cut lung slicesSemantic proximity Read ↗

Functional human iPSC-derived alveolar-like cells cultured in a miniaturized 96‑Transwell air-liquid interface model.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.