Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genetic influences and causal pathways shared between cannabis use disorder and other substance use traits.

Molecular psychiatry - 1 Sept 2024

Galimberti Marco, Levey Daniel F, Deak Joseph D, Zhou Hang, Stein Murray B, Gelernter Joel

Abstract excerpt

Cannabis use disorder (CanUD) has increased with the legalization of the use of cannabis. Around 20% of individuals using cannabis develop CanUD, and the number of users has grown with increasing ease of access. CanUD and other substance use disorders (SUDs) are associated phenotypically and genetically. We leveraged new CanUD genomics data to undertake genetically-informed analyses with unprecedented power, to...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cannabis
Share to Genome-Wide Association Study

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genomics

Share to Marijuana Abuse

Share to Mendelian Randomization Analysis

Share to Opioid-Related Disorders

Share to Substance-Related Disorders

Share to Tobacco Use Disorder

Source

Open in PubMed Open full text

Identifiers

PMID: 38580809
DOI: 10.1038/s41380-024-02548-y
PMCID: PMC11419938

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genetic influences and causal pathways shared between cannabis use disorder and other substance use traits.PMID 38580809Read ↗

A large-scale genome-wide association study meta-analysis of cannabis use disorder.Semantic proximity Read ↗

The genetic aetiology of cannabis use: from twin models to genome-wide association studies and beyond.Semantic proximity Read ↗

Multi-ancestry genome-wide association study of cannabis use disorder yields insight into disease biology and public health implications.Semantic proximity Read ↗

High genes: Genetic underpinnings of cannabis use phenotypes.Semantic proximity Read ↗

The genetics of cannabis lifetime use.Semantic proximity Read ↗

The genetic architecture of substance use and its diverse correlations with mental health traits.Semantic proximity Read ↗

GWAS of lifetime cannabis use reveals new risk loci, genetic overlap with psychiatric traits, and a causal influence of schizophrenia.Semantic proximity Read ↗

Genomic relationships across psychiatric disorders including substance use disorders.Semantic proximity Read ↗

Genetics of cannabis ever-use and frequency across ancestries implicate novel loci and brain-specific biologySemantic proximity Read ↗

Genetics of cannabis use in opioid use disorder: A genome-wide association and polygenic risk score study.Semantic proximity Read ↗

DSM-5 cannabis use disorder: a phenotypic and genomic perspective.Semantic proximity Read ↗

Genome-wide association study in individuals of European and African ancestry and multi-trait analysis of opioid use disorder identifies 19 independent genome-wide significant risk loci.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.