Tumor necrosis factor α-induced protein 8-like-2 controls microglia phenotype via metabolic reprogramming in BV2 microglial cells and responses to neuropathic pain.

doi:10.1016/j.biocel.2024.106541

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Article

Tumor necrosis factor α-induced protein 8-like-2 controls microglia phenotype via metabolic reprogramming in BV2 microglial cells and responses to neuropathic pain.

The international journal of biochemistry & cell biology - 1 Apr 2024

Li Yeqi, Yin Cui, Jiang Jinhong, Yang Huan, Zhang Feifei, Xing Yanhong, Wang Wuyang, Lu Chen

Abstract excerpt

Microglial are major players in neuroinflammation that have recently emerged as potential therapeutic targets for neuropathic pain. Glucose metabolic programming has been linked to differential activation state and function in microglia. Tumor necrosis factor α-induced protein 8-like-2 (TNFAIP8L2) is an important component in regulating the anti-inflammatory response. However, the role of TNFAIP8L2 in microglia...

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