Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

A role for GLUT3 in glioblastoma cell invasion that is not recapitulated by GLUT1

1 Jan 2021

Abstract excerpt

The multifaceted roles of metabolism in invasion have been investigated across many cancers. The brain tumor glioblastoma (GBM) is a highly invasive and metabolically plastic tumor with an inevitable recurrence. The neuronal glucose transporter 3 (GLUT3) was previously reported to correlate with poor glioma patient survival and be upregulated in GBM cells to promote therapeutic resistance and survival under...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer Cells and Metastasis
Share to Cancer, Hypoxia, and Metabolism
Share to Glioma Diagnosis and Treatment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 33843470
DOI: 10.1080/19336918.2021.1903684

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

A role for GLUT3 in glioblastoma cell invasion that is not recapitulated by GLUT1PMID 33843470Read ↗

Microtubule-associated NAV3 regulates invasive phenotypes in glioblastoma cells.Semantic proximity Read ↗

GLUT3 is induced during epithelial-mesenchymal transition and promotes tumor cell proliferation in non-small cell lung cancerSemantic proximity Read ↗

A molecular signature for the G6PC3/SLC37A2/SLC37A4 interactors in glioblastoma disease progression and in the acquisition of a brain cancer stem cell phenotype.Semantic proximity Read ↗

Identification of a Druggable Pathway Controlling Glioblastoma InvasivenessSemantic proximity Read ↗

Identification of Compounds That Decrease Glioblastoma Growth and Glucose Uptake <i>in Vitro</i>Semantic proximity Read ↗

Hexosaminidase B-driven cancer cell-macrophage co-dependency promotes glycolysis addiction and tumorigenesis in glioblastoma.Semantic proximity Read ↗

Gene expression of GLUT3 and GLUT1 glucose transporters in human brain tumorsSemantic proximity Read ↗

Dual roles of HK3 in regulating the network between tumor cells and tumor-associated macrophages in neuroblastomaSemantic proximity Read ↗

Membrane Proteome Analysis of Glioblastoma Cell InvasionSemantic proximity Read ↗

Glucose-6-phosphatase is a key metabolic regulator of glioblastoma invasion.Semantic proximity Read ↗

Glycation Leads to Increased Invasion of Glioblastoma CellsSemantic proximity Read ↗

18F-FDG PET imaging reveals GW4869's suppression of glioblastoma malignant progression and reversal of TMZ chemoresistance via glucose metabolic phenotype remodeling.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.