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Article

Pten and Dicer1 loss in the mouse uterus causes poorly differentiated endometrial adenocarcinoma

25 Aug 2020

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 32843721
DOI: 10.1038/s41388-020-01434-5

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Related research

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Pten and Dicer1 loss in the mouse uterus causes poorly differentiated endometrial adenocarcinomaPMID 32843721Read ↗

<i>Pten</i> and <i>Dicer1</i> loss causes poorly-differentiated endometrial adenocarcinoma in miceSemantic proximity Read ↗

Genetic analyses reveal a requirement for Dicer1 in the mouse urogenital tractSemantic proximity Read ↗

Biallelic Dicer1 Mutations in the Gynecologic Tract of Mice Drive Lineage-Specific Development of DICER1 Syndrome-Associated Cancer.Semantic proximity Read ↗

Inactivation of Dicer1 has a severe cumulative impact on the formation of mature germ cells in mouse testesSemantic proximity Read ↗

Ovarian insufficiency and CTNNB1 mutations drive malignant transformation of endometrial hyperplasia with altered PTEN/PI3K activitiesSemantic proximity Read ↗

PI3K/PTEN/AKT Genetic Mouse Models of Endometrial Carcinoma.Semantic proximity Read ↗

Human Cripto-1 overexpression in the mouse mammary gland results in the development of hyperplasia and adenocarcinomaSemantic proximity Read ↗

Conditional loss of uterine Pten unfailingly and rapidly induces endometrial cancer in mice.Semantic proximity Read ↗

Disruption of Dicer1 Induces Dysregulated Fetal Gene Expression and Promotes HepatocarcinogenesisSemantic proximity Read ↗

Pten is essential for embryonic development and tumour suppressionSemantic proximity Read ↗

Generation and characterization of a mouse model of conditional <i>Chd4</i> knockout in the endometrial epitheliumSemantic proximity Read ↗

The uterine epithelial loss of Pten is inefficient to induce endometrial cancer with intact stromal Pten.Semantic proximity Read ↗

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