Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

l-Theanine attenuates neointimal hyperplasia via suppression of vascular smooth muscle cell phenotypic modulation.

The Journal of nutritional biochemistry - 1 Aug 2020

Bi Aijing, Hang Qiyun, Huang Youfang, Zheng Shuangning, Bi Xiaowen, Zhang Zhengping, Yin Zhimin, Luo Lan

Abstract excerpt

Neointimal hyperplasia is a prominent pathological phenomenon in the process of stent restenosis. Abnormal proliferation and migration of vascular smooth muscle cells (VSMCs) play major pathological processes involved in the development of restenosis. l-Theanine, one of the major amino acid components in green tea, has been reported to improve vascular function. Here we display the effects of l-theanine on...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Becaplermin
Share to Carotid Artery Injuries

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Coronary Restenosis

Share to ets-Domain Protein Elk-1

Share to Glutamates

Share to Mitogen-Activated Protein Kinase 1

Share to Neointima

Source

Identifiers

PMID: 32402912
DOI: 10.1016/j.jnutbio.2020.108398

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

l-Theanine attenuates neointimal hyperplasia via suppression of vascular smooth muscle cell phenotypic modulation.PMID 32402912Read ↗

Ginsenoside Rb₁ inhibits the carotid neointimal hyperplasia induced by balloon injury in rats via suppressing the phenotype modulation of vascular smooth muscle cells.Semantic proximity Read ↗

Lycorine hydrochloride reveals a novel role in preventing neointimal hyperplasia through MAPK-mediated phenotypic switching of smooth muscle cells.Semantic proximity Read ↗

Colchicine reduces neointima formation and VSMC phenotype transition by modulating SRF-MYOCD activation and autophagy.Semantic proximity Read ↗

The novel piperlongumine-ligustrazine derivative PL-Lig-11 inhibits VSMC phenotypic switching and intimal hyperplasia via the PDGFRα/β-RhoGDI3-NLRP3 inflammasome axis.Semantic proximity Read ↗

Tanshinone II A attenuates vascular remodeling through klf4 mediated smooth muscle cell phenotypic switching.Semantic proximity Read ↗

Piperlonguminine attenuates atherosclerosis in mice via phenotype switching inhibition of vascular smooth muscle cells.Semantic proximity Read ↗

A newly synthesized Ligustrazine stilbene derivative inhibits PDGF-BB induced vascular smooth muscle cell phenotypic switch and proliferation via delaying cell cycle progression.Semantic proximity Read ↗

miRNA-92a Inhibits the Phenotypic Modulation of Vascular Smooth Muscle Cells and May Help to Prevent In-Stent RestenosisSemantic proximity Read ↗

Euonymine inhibits in-stent restenosis through enhancing contractile phenotype of vascular smooth muscle cells via modulating the PTEN/AKT/mTOR signaling pathway.Semantic proximity Read ↗

miRNA‑92a inhibits vascular smooth muscle cell phenotypic modulation and may help prevent in‑stent restenosis.Semantic proximity Read ↗

Targeting PDK4 attenuates neointimal hyperplasia and regulates VSMC phenotypic switching, apoptosis, and autophagy.Semantic proximity Read ↗

Salidroside inhibits platelet-derived growth factor BB-induced human aortic smooth muscle cell phenotype switching via PDGFR-β/AKT/mTOR/HIF-1α pathway 1Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.