Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

NOD1/NOD2-mediated recognition of non-typeable Haemophilus influenzae activates innate immunity during otitis media

31 Aug 2019

Abstract excerpt

Pathogen recognition following infection in mammals depends mainly on TLRs and NLRs. Herein, we evaluate the role of NOD1 and NOD2 signaling in the inflammatory responses of the middle ear (ME) mucosa and leukocytes recruitment to infection site during otitis media (OM). OM is a common pediatric disease with prevalent repercussions on hearing health. While many risk factors have been implicated to OM proneness,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antimicrobial Peptides and Activities
Share to Ear Surgery and Otitis Media
Share to Immune Response and Inflammation

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 31474163
DOI: 10.1177/1753425919872266

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

NOD1/NOD2-mediated recognition of non-typeable Haemophilus influenzae activates innate immunity during otitis mediaPMID 31474163Read ↗

NOD1 and NOD2 in inflammatory and infectious diseasesSemantic proximity Read ↗

Nod‐like proteins in inflammation and diseaseSemantic proximity Read ↗

Innate Signaling in Otitis Media: Pathogenesis and RecoverySemantic proximity Read ↗

Nod2: The intestinal gate keeperSemantic proximity Read ↗

Nod1 and Nod2 in innate immunity and human inflammatory disorders.Semantic proximity Read ↗

Innate immune recognition of microbes through Nod1 and Nod2: implications for diseaseSemantic proximity Read ↗

Nod-like proteins in immunity, inflammation and diseaseSemantic proximity Read ↗

Innate Immune Sensing of Microbes by Nod ProteinsSemantic proximity Read ↗

Resolving the field: a role for Nod2 in T cells.Semantic proximity Read ↗

TLR4-mediated induction of TLR2 signaling is critical in the pathogenesis and resolution of otitis mediaSemantic proximity Read ↗

NOD-like receptors--pivotal guardians of the immunological integrity of barrier organs.Semantic proximity Read ↗

NOD1 and NOD2 in inflammation, immunity and diseaseSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.