Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Modeling Cardiac Fibrosis in Mice: (Myo)Fibroblast Phenotype After Ischemia.

Methods in molecular biology (Clifton, N.J.) - 1 Jan 2017

Fraccarollo Daniela, Galuppo Paolo, Bauersachs Johann

Abstract excerpt

Cardiac (myo)fibroblasts play a key role in the regulation of wound healing and pathogenic remodeling after myocardial infarction. Impaired scar formation and alterations of the extracellular matrix network precipitate cardiac dysfunction leading to increased morbidity and mortality. Therapeutic approaches addressing (myo)fibroblast phenotype appear to be useful in preventing progressive structural, electrical,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Biomarkers
Share to Cardiomyopathies
Share to Cell Separation

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Fibroblasts

Share to Fibrosis

Share to Flow Cytometry

Share to Ischemia

Share to Myofibroblasts

Source

Identifiers

PMID: 28836199
DOI: 10.1007/978-1-4939-7113-8_9

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Modeling Cardiac Fibrosis in Mice: (Myo)Fibroblast Phenotype After Ischemia.PMID 28836199Read ↗

Consequences of PDGFRα <sup>+</sup> fibroblast reduction in adult murine heartsSemantic proximity Read ↗

Fibroblasts from human postmyocardial infarction scars acquire properties of cardiomyocytes after transduction with a recombinant myocardin gene.Semantic proximity Read ↗

Cardiac Fibrosis and FibroblastsSemantic proximity Read ↗

Gaining myocytes or losing fibroblasts: Challenges in cardiac fibroblast reprogramming for infarct repair.Semantic proximity Read ↗

Specialized fibroblast differentiated states underlie scar formation in the infarcted mouse heartSemantic proximity Read ↗

Fibroblast polarization over the myocardial infarction time continuum shifts roles from inflammation to angiogenesisSemantic proximity Read ↗

Changes in cardiac resident fibroblast physiology and phenotype in aging.Semantic proximity Read ↗

Isolation of Adult Mouse Cardiac FibroblastsSemantic proximity Read ↗

Origins of Cardiac FibroblastsSemantic proximity Read ↗

Cardiac mesenchymal stem cells contribute to scar formation after myocardial infarction.Semantic proximity Read ↗

A novel transgenic Cre allele to label mouse cardiac conduction system.Semantic proximity Read ↗

Adventitial leptin receptor-expressing fibroblasts are preferential contributors to fibrotic remodeling of the heart post infarctionSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.