Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genetic evidence for role of integration of fast and slow neurotransmission in schizophrenia.

Molecular psychiatry - 1 Jun 2017

Devor A, Andreassen O A, Wang Y, Mäki-Marttunen T, Smeland O B, Fan C-C, Schork A J, Holland D, Thompson W K, Witoelar A, Chen C-H, Desikan R S, McEvoy L K, Djurovic S, Greengard P, Svenningsson P, Einevoll G T, Dale A M

Abstract excerpt

The most recent genome-wide association studies (GWAS) of schizophrenia (SCZ) identified hundreds of risk variants potentially implicated in the disease. Further, novel statistical methodology designed for polygenic architecture revealed more potential risk variants. This can provide a link betwe...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Brain
Share to Dopamine
Share to Dopamine and cAMP-Regulated Phosphoprotein 32

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to gamma-Aminobutyric Acid

Share to Multifactorial Inheritance

Share to Receptors, Metabotropic Glutamate

Share to Schizophrenia

Share to Synaptic Transmission

Source

Open in PubMed Open full text

Identifiers

PMID: 28348379
DOI: 10.1038/mp.2017.33
PMCID: PMC5495879

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Prepared record publication-28348379-2026-09-05.1. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genetic evidence for role of integration of fast and slow neurotransmission in schizophrenia.PMID 28348379Read ↗

Genetic risk for schizophrenia: convergence on synaptic pathways involved in plasticity.Semantic proximity Read ↗

A Transcriptome Wide Association Study implicates specific pre- and post-synaptic abnormalities in SchizophreniaSemantic proximity Read ↗

Genetic analysis of schizophrenia and bipolar disorder reveals polygenicity but also suggests new directions for molecular interrogation.Semantic proximity Read ↗

Mapping genomic loci implicates genes and synaptic biology in schizophrenia.Semantic proximity Read ↗

Novel treatment strategies for schizophrenia from improved understanding of genetic risk.Semantic proximity Read ↗

Integrating rare variant genetics and brain transcriptome data implicates novel schizophrenia putative risk genes.Semantic proximity Read ↗

The polygenic architecture of schizophrenia - rethinking pathogenesis and nosology.Semantic proximity Read ↗

Recent genetic findings in schizophrenia and their therapeutic relevance.Semantic proximity Read ↗

Schizophrenia Genomics: Convergence on Synaptic Development, Adult Synaptic Plasticity, or Both?Semantic proximity Read ↗

Enhancing genetic discovery through narrow phenotyping in schizophrenia.Semantic proximity Read ↗

Convergent functional genomics of schizophrenia: from comprehensive understanding to genetic risk prediction.Semantic proximity Read ↗

Comprehensive pathway analyses of schizophrenia risk loci point to dysfunctional postsynaptic signalingSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.