Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

DNA base excision repair genetic risk scores, oxidative balance, and incident, sporadic colorectal adenoma.

Molecular carcinogenesis - 1 Jun 2017

Wang Tengteng, Goodman Michael, Sun Yan V, Thyagarajan Bharat, Gross Myron, Bostick Roberd M

Abstract excerpt

Associations of individual base excision repair (BER) genotypes with colorectal adenoma risk are unclear, but likely modest. However, genetic risk scores (GRS) that aggregate information from multiple genetic variants might be useful for assessing genetic predisposition to colorectal adenoma. We analyzed data pooled from three colonoscopy-based case-control studies of incident, sporadic colorectal adenoma...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adenoma
Share to Colon
Share to Colorectal Neoplasms

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to DNA Repair

Share to Odds Ratio

Share to Oxidation-Reduction

Share to Oxidative Stress

Share to Rectum

Source

Identifiers

PMID: 28120344
DOI: 10.1002/mc.22620

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

DNA base excision repair genetic risk scores, oxidative balance, and incident, sporadic colorectal adenoma.PMID 28120344Read ↗

Associations of oxidative balance-related exposures with incident, sporadic colorectal adenoma according to antioxidant enzyme genotypes.Semantic proximity Read ↗

Genetic variation in the base excision repair pathway, environmental risk factors, and colorectal adenoma risk.Semantic proximity Read ↗

Bulky DNA adduct levels in normal-appearing colon mucosa, and the prevalence of colorectal adenomas.Semantic proximity Read ↗

Genetic Polymorphisms of DNA Repair Pathways in Sporadic Colorectal CarcinogenesisSemantic proximity Read ↗

Individual and Joint Associations of Genetic Risk and Healthy Lifestyle Score with Colorectal Neoplasms Among Participants of Screening Colonoscopy.Semantic proximity Read ↗

Characterization of Additive Gene-environment Interactions For Colorectal Cancer Risk.Semantic proximity Read ↗

Genetic Risk Score, Combined Lifestyle Factors and Risk of Colorectal CancerSemantic proximity Read ↗

Genetic variation in base excision repair genes and the prevalence of advanced colorectal adenoma.Semantic proximity Read ↗

Germline susceptibility to colorectal cancer due to base-excision repair gene defects.Semantic proximity Read ↗

No evidence that associations of incident, sporadic colorectal adenoma with its major modifiable risk factors differ by chromosome 8q24 region rs6983267 genotype.Semantic proximity Read ↗

DNA repair gene polymorphisms and tobacco smoking in the risk for colorectal adenomas.Semantic proximity Read ↗

Genetic Risk Score Is Associated With Prevalence of Advanced Neoplasms in a Colorectal Cancer Screening Population.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.