Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

IL-10 differentially controls the infiltration of inflammatory macrophages and antigen-presenting cells during inflammation.

European journal of immunology - 1 Sept 2016

Liao Chia-Te, Rosas Marcela, Davies Luke C, Giles Peter J, Tyrrell Victoria J, O'Donnell Valerie B, Topley Nicholas, Humphreys Ian R, Fraser Donald J, Jones Simon A, Taylor Philip R

Abstract excerpt

The inflammatory activation and recruitment of defined myeloid populations is essential for controlling the bridge between innate and adaptive immunity and shaping the immune response to microbial challenge. However, these cells exhibit significant functional heterogeneity and the inflammatory signals that differentially influence their effector characteristics are poorly characterized. In this study, we defined...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antigen Presentation
Share to Antigen-Presenting Cells

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Dendritic Cells

Share to Immunomodulation

Share to Immunophenotyping

Share to Interleukin-10

Share to Peritonitis

Share to Receptors, CCR2

Source

Open in PubMed Open full text

Identifiers

PMID: 27378515
DOI: 10.1002/eji.201646528
PMCID: PMC5026061

Genes

APC

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

IL-10 differentially controls the infiltration of inflammatory macrophages and antigen-presenting cells during inflammation.PMID 27378515Read ↗

Human intestinal macrophages display profound inflammatory anergy despite avid phagocytic and bacteriocidal activity.Semantic proximity Read ↗

Pattern recognition receptors and differentiation antigens define murine myeloid cell heterogeneity ex vivo.Semantic proximity Read ↗

Myeloid arginase-1 controls excessive inflammation and modulates T cell responses in Pseudomonas aeruginosa pneumonia.Semantic proximity Read ↗

Macrophage phenotype in the subclinical gut inflammation of patients with ankylosing spondylitis.Semantic proximity Read ↗

Regulation of the IL-10-driven macrophage phenotype under incoherent stimuli.Semantic proximity Read ↗

Serum level of IL-10 is increased in patients with endometriosis, and IL-10 promotes the growth of lesions in a murine model.Semantic proximity Read ↗

Ongoing Exposure to Peritoneal Dialysis Fluid Alters Resident Peritoneal Macrophage Phenotype and Activation Propensity.Semantic proximity Read ↗

The Phenotype of Inflammatory Macrophages Is Stimulus Dependent: Implications for the Nature of the Inflammatory ResponseSemantic proximity Read ↗

Ongoing exposure to peritoneal dialysis fluid alters resident peritoneal macrophage phenotype and activation propensitySemantic proximity Read ↗

IL-10 distinguishes a unique population of activated, effector-like CD8+ T cells in murine acute liver inflammation.Semantic proximity Read ↗

Calprotectin expression and mononuclear phagocyte subpopulations in peripheral blood and bronchoalveolar lavage.Semantic proximity Read ↗

Human CD1c (BDCA-1)+ myeloid dendritic cells secrete IL-10 and display an immuno-regulatory phenotype and function in response to Escherichia coli.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.