Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

STAT3 is Overactivated in Gastric Cancer Stem-Like Cells.

1 Jan 2016

Abstract excerpt

OBJECTIVE: Gastric cancer (GC) is widely associated with chronic inflammation. The pro inflammatory microenvironment provides conditions that disrupt stem/progenitor cell proliferation and differentiation. The signal transducer and activator of transcrip- tion-3 (STAT3) signaling pathway is involved in inflammation and also contributes to the maintenance of embryonic stem cell (ESCs) pluripotency. Here, we have...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer Cells and Metastasis
Share to Cancer, Stress, Anesthesia, and Immune Response
Share to Cytokine Signaling Pathways and Interactions

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 26862521
DOI: 10.22074/cellj.2016.3834

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

STAT3 is Overactivated in Gastric Cancer Stem-Like Cells.PMID 26862521Read ↗

STAT3 Pathway in Gastric Cancer: Signaling, Therapeutic Targeting and Future ProspectsSemantic proximity Read ↗

Spheroid body-forming cells in the human gastric cancer cell line MKN-45 possess cancer stem cell propertiesSemantic proximity Read ↗

STAT3 is involved in esophageal carcinogenesis through regulation of Oct-1Semantic proximity Read ↗

STAT3 signaling pathway is necessary for cell survival and tumorsphere forming capacity in ALDH+/CD133+ stem cell-like human colon cancer cellsSemantic proximity Read ↗

Induction, growth, drug resistance, and metastasis: A comprehensive summary of the relationship between STAT3 and gastric cancerSemantic proximity Read ↗

The promotion of the transformation of quiescent gastric cancer stem cells by IL-17 and the underlying mechanismsSemantic proximity Read ↗

Establishment and characterization of chemotherapy-enriched sphere-forming cells with stemness phenotypes as a new cell line (BAG50) of gastric carcinoma.Semantic proximity Read ↗

Molecular mechanism, regulation, and therapeutic targeting of the STAT3 signaling pathway in esophageal cancer (Review)Semantic proximity Read ↗

DDIT3 modulates cancer stemness in gastric cancer by directly regulating CEBPβSemantic proximity Read ↗

Transcription Factor STAT3 as a Novel Molecular Target for Cancer PreventionSemantic proximity Read ↗

Role of STAT3 in cancer cell epithelial‑mesenchymal transition (Review)Semantic proximity Read ↗

Stem cells in gastric cancer.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.