Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

ADDing a piece to the puzzle of cognition in schizophrenia.

European journal of medical genetics - 1 Jan 2016

Bosia Marta, Pigoni Alessandro, Zagato Laura, Merlino Lino, Casamassima Nunzia, Lorenzi Cristina, Pirovano Adele, Smeraldi Enrico, Manunta Paolo, Cavallaro Roberto

Abstract excerpt

The biological bases of cognitive impairment in schizophrenia are poorly understood and may lie in insults in neurodevelopment, leading to alterations in critical structures. Synapses proteins are claimed to have etiopathogenic roles and more direct effects on core cognitive functions. Adducins family proteins seem of great interest, as they are fundamental constituents of synapses, involved in actin cytoskeleton...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cognition Disorders
Share to Cytoskeletal Proteins

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to DNA Mutational Analysis

Share to ADD1 gene

Share to Polymorphism, Single Nucleotide

Share to Schizophrenia

Source

Identifiers

PMID: 26723519
DOI: 10.1016/j.ejmg.2015.12.012

Genes

ADD2
ADD1
ADD3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

ADDing a piece to the puzzle of cognition in schizophrenia.PMID 26723519Read ↗

[Results and promises of genetics of cognitive impairment in schizophrenia: molecular-genetic approaches].Semantic proximity Read ↗

A molecular pathway analysis stresses the role of inflammation and oxidative stress towards cognition in schizophrenia.Semantic proximity Read ↗

NRG3 contributes to cognitive deficits in chronic patients with schizophrenia.Semantic proximity Read ↗

Cognitive Characterization of Schizophrenia Risk Variants Involved in Synaptic Transmission: Evidence of CACNA1C's Role in Working Memory.Semantic proximity Read ↗

The genetic variations in SAP97 gene and the risk of schizophrenia in the Chinese Han population: a further study.Semantic proximity Read ↗

Evidence that a working memory cognitive phenotype within schizophrenia has a unique underlying biology.Semantic proximity Read ↗

Translating advances in the molecular basis of schizophrenia into novel cognitive treatment strategies.Semantic proximity Read ↗

Association between cognitive impairment and apolipoprotein A1 or apolipoprotein B levels is regulated by apolipoprotein E variant rs429358 in patients with chronic schizophrenia.Semantic proximity Read ↗

Polygenic risk score, genome-wide association, and gene set analyses of cognitive domain deficits in schizophrenia.Semantic proximity Read ↗

A promoter variant of SHANK1 affects auditory working memory in schizophrenia patients and in subjects clinically at risk for psychosis.Semantic proximity Read ↗

Genome-wide association study detected novel susceptibility genes for social cognition impairment in people with schizophrenia.Semantic proximity Read ↗

Brain-derived neurotrophic factor (BDNF) and neurocognitive deficits in people with schizophrenia: a meta-analysis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.