Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Toll-like receptor 3 gene polymorphisms are not associated with the risk of hepatitis B and hepatitis C virus infection.

Revista da Sociedade Brasileira de Medicina Tropical - 1 Jan 2000

Sá Keyla Santos Guedes de, Pires-Neto Orlando de Souza, Santana Barbara Brasil, Gomes Samara Tatielle Monteiro, Amoras Ednelza da Silva Graça, Conde Simone Regina da Silva, Demachki Sâmia, Azevedo Vânia Nakauth, Machado Luiz Fernando Almeida, Martins-Feitosa Rosimar Neris, Ishak Marluísa de Oliveira Guimarães, Ishak Ricardo, Vallinoto Antonio Carlos Rosário

Abstract excerpt

INTRODUCTION: The present study investigated the prevalence of two single-nucleotide polymorphisms (SNPs) in the Toll-like receptor 3 (TLR3) gene in patients infected with hepatitis B virus (HBV) and hepatitis C virus (HCV). METHODS: Samples collected from HCV (n = 74) and HBV (n = 35) carriers were subjected to quantitative real-time PCR (qPCR) to detect the presence of the SNPs rs5743305 and rs3775291 in TLR3...

Read the complete abstract on PubMed

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alanine Transaminase
Share to Aspartate Aminotransferases
Share to Disease Progression
Share to gamma-Glutamyltransferase
Share to Haplotypes
Share to Hepatitis B, Chronic
Share to Hepatitis C, Chronic
Share to Toll-Like Receptor 3

Source

Identifiers

PMID: 25992926
DOI: 10.1590/0037-8682-0008-2015

Genes

TLR3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Toll-like receptor 3 gene polymorphisms are not associated with the risk of hepatitis B and hepatitis C virus infection.PMID 25992926Read ↗

TLR3 gene polymorphisms and liver disease manifestations in chronic hepatitis C.Semantic proximity Read ↗

Toll-Like Receptor 3 is Associated With the Risk of HCV Infection and HBV-Related Diseases.Semantic proximity Read ↗

Toll-like receptor 3 polymorphism and its association with hepatitis B virus infection in Saudi Arabian patients.Semantic proximity Read ↗

Toll Like Receptor 3 Polymorphisms in Hepatitis B Virus Infection.Semantic proximity Read ↗

Association of Toll-Like Receptor 3 Single-Nucleotide Polymorphisms and Hepatitis C Virus Infection.Semantic proximity Read ↗

Lack of Association between Polymorphisms of the TLR4 Gene and Infection with the Hepatitis B and C Viruses.Semantic proximity Read ↗

Toll-like receptor polymorphisms are not associated with liver cirrhosis in hepatitis B virus infected Korean patients.Semantic proximity Read ↗

Genetic polymorphisms in Toll-like receptor 3 gene are associated with the risk of hepatitis B virus-related liver diseases in a Chinese population.Semantic proximity Read ↗

Association of Toll-like receptor 3 polymorphisms with chronic hepatitis B and hepatitis B-related acute-on-chronic liver failure.Semantic proximity Read ↗

Association of Toll-Like Receptor 3 and Toll-Like Receptor 9 Single Nucleotide Polymorphisms with Hepatitis C Virus Infection and Hepatic Fibrosis in Egyptian Patients.Semantic proximity Read ↗

TLR3 and TLR4 SNP variants in the liver disease resulting from hepatitis B virus and hepatitis C virus infectionSemantic proximity Read ↗

Study of Toll-like Receptor 3 Gene Polymorphism as a Novel Risk Factor for HCV-related Hepatocellular Carcinoma in Egypt.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.