Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Association of Toll-Like Receptor 3 Single-Nucleotide Polymorphisms and Hepatitis C Virus Infection.

Journal of immunology research - 1 Jan 2017

Al-Anazi Mashael R, Matou-Nasri Sabine, Abdo Ayman A, Sanai Faisal M, Alkahtani Saad, Alarifi Saud, Alkahtane Abdullah A, Al-Yahya Hamad, Ali Daoud, Alessia Mohammed S, Alshahrani Bushra, Al-Ahdal Mohammed N, Al-Qahtani Ahmed A

Abstract excerpt

Toll-like receptor 3 (TLR3) plays a key role in innate immunity by recognizing pathogenic, double-stranded RNAs. Thus, activation of TLR3 is a major factor in antiviral defense and tumor eradication. Although downregulation of TLR3 gene expression has been mainly reported in patients infected with hepatitis C virus (HCV), the influence of TLR3 genotype on the risk of HCV infection, HCV-related cirrhosis, and/or...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Carcinoma, Hepatocellular
Share to Fibrosis

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Genetic Association Studies

Share to Hepatitis C, Chronic

Share to Liver Neoplasms

Share to Saudi Arabia

Share to Toll-Like Receptor 3

Source

Open in PubMed Open full text

Identifiers

PMID: 28127569
DOI: 10.1155/2017/1590653
PMCID: PMC5239973

Genes

TLR3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Association of Toll-Like Receptor 3 Single-Nucleotide Polymorphisms and Hepatitis C Virus Infection.PMID 28127569Read ↗

Toll-like receptor 3 polymorphism and its association with hepatitis B virus infection in Saudi Arabian patients.Semantic proximity Read ↗

Toll-Like Receptor 3 is Associated With the Risk of HCV Infection and HBV-Related Diseases.Semantic proximity Read ↗

The association of single nucleotide polymorphisms of Toll-like receptor 3, Toll-like receptor 7 and Toll-like receptor 8 genes with the susceptibility to HCV infection.Semantic proximity Read ↗

The association of toll-like receptor 4 polymorphism with hepatitis C virus infection in Saudi Arabian patients.Semantic proximity Read ↗

Study of Toll-like Receptor 3 Gene Polymorphism as a Novel Risk Factor for HCV-related Hepatocellular Carcinoma in Egypt.Semantic proximity Read ↗

Toll Like Receptor 3 Polymorphisms in Hepatitis B Virus Infection.Semantic proximity Read ↗

Genetic polymorphisms in Toll-like receptor 3 gene are associated with the risk of hepatitis B virus-related liver diseases in a Chinese population.Semantic proximity Read ↗

Association of Toll-Like Receptor 3 and Toll-Like Receptor 9 Single Nucleotide Polymorphisms with Hepatitis C Virus Infection and Hepatic Fibrosis in Egyptian Patients.Semantic proximity Read ↗

TLR3 gene polymorphisms and liver disease manifestations in chronic hepatitis C.Semantic proximity Read ↗

Genetic Polymorphism in Toll-Like Receptor 3 and Interferon Regulatory Factor 3 in Hepatitis C Virus-Infected Patients: Correlation with Liver Cirrhosis.Semantic proximity Read ↗

Association between a functional polymorphism in Toll-like receptor 3 and chronic hepatitis C in liver transplant recipients.Semantic proximity Read ↗

Association of Toll-like receptor 3 and Toll-like receptor 9 single-nucleotide polymorphisms with hepatitis C virus persistence among Egyptians.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.