Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Selection of dinB alleles suppressing survival loss upon dinB overexpression in Escherichia coli.

Journal of bacteriology - 15 Aug 2014

Benson Ryan W, Cafarelli Tiziana M, Rands Thomas J, Lin Ida, Godoy Veronica G

Abstract excerpt

Escherichia coli strains overproducing DinB undergo survival loss; however, the mechanisms regulating this phenotype are poorly understood. Here we report a genetic selection revealing DinB residues essential to effect this loss-of-survival phenotype. The selection uses strains carrying both an a...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Escherichia coli Proteins
Share to Microbial Viability
Share to Mutagenesis, Insertional

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Mutant Proteins

Share to Plasmids

Share to Point Mutation

Share to Sequence Deletion

Share to Suppression, Genetic

Source

Open in PubMed Open full text

Identifiers

PMID: 24914188
DOI: 10.1128/JB.01782-14
PMCID: PMC4135642

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-24914188-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Selection of dinB alleles suppressing survival loss upon dinB overexpression in Escherichia coli.PMID 24914188Read ↗

DinB upregulation is the sole role of the SOS response in stress-induced mutagenesis in Escherichia coli.Semantic proximity Read ↗

Overproduction of Escherichia coli DNA polymerase DinB (Pol IV) inhibits replication fork progression and is lethal.Semantic proximity Read ↗

The dinB operon and spontaneous mutation in Escherichia coli.Semantic proximity Read ↗

A role for nonessential domain II of initiator protein, DnaA, in replication control.Semantic proximity Read ↗

Novel Escherichia coli active site dnaE alleles with altered base and sugar selectivity.Semantic proximity Read ↗

An active site aromatic triad in Escherichia coli DNA Pol IV coordinates cell survival and mutagenesis in different DNA damaging agents.Semantic proximity Read ↗

What limits the efficiency of double-strand break-dependent stress-induced mutation in Escherichia coli?Semantic proximity Read ↗

Mutant DnaAs of Escherichia coli that are refractory to negative control.Semantic proximity Read ↗

A ΔdinB mutation that sensitizes Escherichia coli to the lethal effects of UV- and X-radiation.Semantic proximity Read ↗

Selection-Enhanced Mutagenesis of lac Genes Is Due to Their Coamplification with dinB Encoding an Error-Prone DNA Polymerase.Semantic proximity Read ↗

SOS mutator DNA polymerase IV functions in adaptive mutation and not adaptive amplification.Semantic proximity Read ↗

The Mutant βE202K Sliding Clamp Protein Impairs DNA Polymerase III Replication Activity.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.