Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

MicroRNA-18a Improves Human Cerebral Arteriovenous Malformation Endothelial Cell Function

8 Nov 2013

Abstract excerpt

BACKGROUND AND PURPOSE: Cerebral arteriovenous malformation (AVM) is a vascular disease that disrupts normal blood flow and leads to serious neurological impairment or death. Aberrant functions of AVM-derived brain endothelial cells (AVM-BECs) are a disease hallmark. Our aim was to use microRNA-18a (miR-18a) as a therapeutic agent to improve AVM-BEC function. METHODS: Human AVM-BECs were tested for growth factor...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Intracranial Aneurysms: Treatment and Complications
Share to Moyamoya disease diagnosis and treatment
Share to Vascular Malformations Diagnosis and Treatment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 24203843
DOI: 10.1161/strokeaha.113.003578

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

MicroRNA-18a Improves Human Cerebral Arteriovenous Malformation Endothelial Cell FunctionPMID 24203843Read ↗

Thrombospondin-1 modulates the angiogenic phenotype of human cerebral arteriovenous malformation endothelial cells.Semantic proximity Read ↗

Aberrant angiogenic characteristics of human brain arteriovenous malformation endothelial cells.Semantic proximity Read ↗

Understanding the pathogenesis of brain arteriovenous malformation: genetic variations, epigenetics, signaling pathways, and immune inflammation.Semantic proximity Read ↗

RNA-Sequencing Highlights Inflammation and Impaired Integrity of the Vascular Wall in Brain Arteriovenous MalformationsSemantic proximity Read ↗

Evidence for endothelial‐to‐mesenchymal transition in human brain arteriovenous malformationsSemantic proximity Read ↗

N6-methyladenosine methyltransferase METTL3 affects the phenotype of cerebral arteriovenous malformation via modulating Notch signaling pathway.Semantic proximity Read ↗

An Insight into the microRNAs Associated with Arteriovenous and Cavernous Malformations of the BrainSemantic proximity Read ↗

Brain arteriovenous malformations.Semantic proximity Read ↗

Advances in the Genetics and Molecular Biology of Brain Arteriovenous Malformations.Semantic proximity Read ↗

Development and characterization of a human model of arteriovenous malformation (AVM)-on-a-chipSemantic proximity Read ↗

Review of treatment and therapeutic targets in brain arteriovenous malformation.Semantic proximity Read ↗

Somatic BrafV600E mutation in the cerebral endothelium induces brain arteriovenous malformations.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.