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Article

Exposure to environmental tobacco smoke, human E-cadherin C-160A polymorphism, and childhood asthma.

Annals of allergy, asthma & immunology : official publication of the American College of Allergy, Asthma, & Immunology - 1 Oct 2013

Wang Ming-Fuu, Kuo Sheng-Hung, Huang Chun-Huang, Chen Yi-Ju, Lin Shin-Hua, Lee Chiung-Ju, Lue Ko-Huang, Wu Shao-Chun, Cho Chung-Yen, Wong Ruey-Hong

Abstract excerpt

BACKGROUND: Environmental tobacco smoke (ETS) is a risk factor for asthma. Importantly, cigarette smoke can decrease the adherence of epithelial cells and increase detachment. The adhesion molecule E-cadherin (CDH1) has an essential role in the formation of epithelial junction. Turnover of the extracellular matrix, which is characterized by airway remodeling, depends on the imbalance between matrix...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Source

Identifiers

PMID: 24054361
DOI: 10.1016/j.anai.2013.07.008

Genes

CDH1

Variants

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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