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Article

No contribution of IP3-R(2) to disease phenotype in models of dilated cardiomyopathy or pressure overload hypertrophy.

Circulation. Heart failure - 1 Mar 2013

Cooley Nicola, Ouyang Kunfu, McMullen Julie R, Kiriazis Helen, Sheikh Farah, Wu Wei, Mu Yongxin, Du Xiao-Jun, Chen Ju, Woodcock Elizabeth A

Abstract excerpt

BACKGROUND: We investigated the contribution of inositol(1,4,5)-trisphosphate (Ins(1,4,5)P3 [IP3]) receptors (IP3-R) to disease progression in mouse models of dilated cardiomyopathy (DCM) and pressure overload hypertrophy. Mice expressing mammalian sterile 20-like kinase and dominant-negative pho...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Source

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Identifiers

PMID: 23258573
DOI: 10.1161/CIRCHEARTFAILURE.112.972158
PMCID: PMC4028972

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-23258573-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

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