Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

DNA methylation at differentially methylated regions of imprinted genes is resistant to developmental programming by maternal nutrition

12 Sept 2012

Abstract excerpt

The nutritional environment in which the mammalian fetus or infant develop is recognized as influencing the risk of chronic diseases, such as type 2 diabetes and hypertension, in a phenomenon that has become known as developmental programming. The late onset of such diseases in response to earlier transient experiences has led to the suggestion that developmental programming may have an epigenetic component,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Birth, Development, and Health
Share to Epigenetics and DNA Methylation
Share to Genetic Syndromes and Imprinting

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 22968513
DOI: 10.4161/epi.22141

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

DNA methylation at differentially methylated regions of imprinted genes is resistant to developmental programming by maternal nutritionPMID 22968513Read ↗

Genomic imprinting, growth control and the allocation of nutritional resources: consequences for postnatal life.Semantic proximity Read ↗

Genome-Wide Methylation and Gene Expression Changes in Newborn Rats following Maternal Protein Restriction and Reversal by Folic AcidSemantic proximity Read ↗

Intrauterine calorie restriction affects placental DNA methylation and gene expressionSemantic proximity Read ↗

Advanced aging phenotype is revealed by epigenetic modifications in rat liver after in utero malnutrition.Semantic proximity Read ↗

Imprinting of Genes and the Barker HypothesisSemantic proximity Read ↗

Induction of altered epigenetic regulation of the hepatic glucocorticoid receptor in the offspring of rats fed a protein-restricted diet during pregnancy suggests that reduced DNA methyltransferase-1 expression is involved in impaired DNA methylation and changes in histone modificationsSemantic proximity Read ↗

Visualizing Changes in Cdkn1c Expression Links Early-Life Adversity to Imprint Mis-regulation in Adults.Semantic proximity Read ↗

Post-weaning diet affects genomic imprinting at the insulin-like growth factor 2 (Igf2) locus.Semantic proximity Read ↗

Maternal Protein Restriction Elevates Cholesterol in Adult Rat Offspring Due to Repressive Changes in Histone Modifications at the Cholesterol 7α-Hydroxylase PromoterSemantic proximity Read ↗

Maternal Diet during Pregnancy Induces Gene Expression and DNA Methylation Changes in Fetal Tissues in SheepSemantic proximity Read ↗

DNA methylation, insulin resistance, and blood pressure in offspring determined by maternal periconceptional B vitamin and methionine statusSemantic proximity Read ↗

Early-life nutrition modulates the epigenetic state of specific rDNA genetic variants in mice.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.