Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Complementation analysis of mutants defective in different steps of HBV reverse transcription.

Gastroenterologia Japonica - 1 Sept 1990

Schaller H, Radziwill G

Abstract excerpt

The HBV P gene encodes a multifunctional polyprotein which contains the major enzymatic activities required for hepadnaviral reverse transcription (protein primer, DNA polymerase, and RNase H). Mutant HBV genomes unable to synthesize fully functional P gene products were analysed for their potential to be rescued by a second mutant HBV genome that either contained a wild type P gene (intergenic complementation)...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Defective Viruses
Share to Genes, Viral
Share to Genetic Complementation Test

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Hepatitis B virus

Share to Mutation

Share to Transcription, Genetic

Share to Viral Proteins

Share to Virus Replication

Source

Identifiers

PMID: 2227268
DOI: 10.1007/BF02779921

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Complementation analysis of mutants defective in different steps of HBV reverse transcription.PMID 2227268Read ↗

Trans-complementation among naturally occurring deletion mutants of hepatitis B virus and integrated viral DNA for the production of viral particles with mutant genomes in hepatoma cell lines.Semantic proximity Read ↗

trans-Complementation of HBV rtM204I mutant replication by HBV wild-type polymerase.Semantic proximity Read ↗

Mutational analysis of the hepatitis B virus P gene product: domain structure and RNase H activity.Semantic proximity Read ↗

Efficient Rescue of Hepatitis C Virus RNA Replication by trans -Complementation with Nonstructural Protein 5ASemantic proximity Read ↗

Naturally Occurring Hepatitis C Virus Subgenomic Deletion Mutants Replicate Efficiently in Huh-7 Cells and Are trans -Packaged In Vitro To Generate Infectious Defective ParticlesSemantic proximity Read ↗

Co-replication analyses of naturally occurring defective hepatitis B virus variants with wild-type.Semantic proximity Read ↗

Intragenic complementation of hepatitis C virus NS5A RNA replication-defective alleles.Semantic proximity Read ↗

Characterization of Determinants Important for Hepatitis C Virus p7 Function in Morphogenesis by Using trans -ComplementationSemantic proximity Read ↗

An efficient trans complementation system for in vivo replication of defective poliovirus mutants.Semantic proximity Read ↗

Hepatitis C virus envelope glycoproteins complementation patterns and the role of the ecto- and transmembrane domains.Semantic proximity Read ↗

Intragenomic complementation of a 3AB mutant in dicistronic polioviruses.Semantic proximity Read ↗

Complementation Analysis of the Flavivirus Kunjin NS3 and NS5 Proteins Defines the Minimal Regions Essential for Formation of a Replication Complex and Shows a Requirement of NS3 in cis for Virus AssemblySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.